W Keeble
Impact in
- Immunology and Allergy top 10%
- Cell Adhesion Molecules Research
- Hematology top 10%
- Hematopoietic Stem Cell Transplantation
Papers in
-
- DNA Repair Mechanisms 3
- TGF-β signaling in diseases 1
-
- Platelet Disorders and Treatments 3
- Hematopoietic Stem Cell Transplantation 2
- Co-authors
- MC Heinrich (3 shared papers)M. Shoyab (1 shared paper)Paul W. Cook (1 shared paper)Mark R. Pittelkow (1 shared paper)Gary D. Shipley (1 shared paper)John P. Adelman (1 shared paper)Gregory D. Plowman (1 shared paper)David Kabat (3 shared papers)
- Journals
- Blood (4 papers)Journal of Clinical Investigation (1 paper)Molecular and Cellular Biology (1 paper)PubMed (2 papers)
- Partner nations
- United States
In The Last Decade
W Keeble
8 papers receiving 472 citations
Peers
Comparison fields: 5 of 57
- Immunology and Allergy 64
- Hematology 105
- Immunology 137
- Oncology 142
- Cell Biology 85
Countries citing papers authored by W Keeble
This map shows the geographic impact of W Keeble's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W Keeble with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W Keeble more than expected).
Fields of papers citing papers by W Keeble
This network shows the impact of papers produced by W Keeble. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W Keeble. The network helps show where W Keeble may publish in the future.
Co-authors
The 19 scholars most cited alongside W Keeble, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1991 | 212 | |
| 2 | 1993 | 116 | |
| 3 | 1995 | 63 | |
| 4 | 1994 | 37 | |
| 5 | 1996 | 16 | |
| 6 | Constitutive and induced expression of hematopoietic growth factor genes by fibroblasts from children with Fanconi anemia. | 1993 | 15 |
| 7 | 1993 | 12 | |
| 8 | Expression of the Fanconi anemia group C gene in hematopoietic cells is not influenced by oxidative stress, cross-linking agents, radiation, heat, or mitotic inhibitory factors. | 1998 | 10 |
About W Keeble
W Keeble is a scholar working on Molecular Biology, Hematology, Immunology, Cell Biology and Physiology, having authored 8 papers that have together received 481 indexed citations. Recurring topics across this work include Platelet Disorders and Treatments (3 papers), DNA Repair Mechanisms (3 papers), Mast cells and histamine (2 papers), Hematopoietic Stem Cell Transplantation (2 papers), T-cell and B-cell Immunology (2 papers), TGF-β signaling in diseases (1 paper), Skin and Cellular Biology Research (1 paper) and Cell Adhesion Molecules Research (1 paper). The work is most often cited by research in Immunology and Allergy (64 citations), Hematology (105 citations), Immunology (137 citations), Oncology (142 citations) and Cell Biology (85 citations). W Keeble has collaborated with scholars based in United States. Frequent co-authors include MC Heinrich, M. Shoyab, Paul W. Cook, Mark R. Pittelkow, Gary D. Shipley, John P. Adelman, Gregory D. Plowman, David Kabat, Michael C. Heinrich and T. D. SMITH. Their work appears in journals such as Blood, Journal of Clinical Investigation, Molecular and Cellular Biology and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.