Valdemar Máximo

79 papers receiving 4.0k citations

Valdemar Máximo's Hit Papers

BRAF mutations and RET/PTC rearrangements are alternative events in the etiopathogenesis of PTC 2003 · 533 citations
5330+7+15Years since publication100200300400500

Peers

Valdemar Máximo
Comparison fields: 5 of 121
  • Endocrinology, Diabetes and Metabolism 2.0k
  • Cancer Research 753
  • Oncology 1.1k
  • Pathology and Forensic Medicine 512
  • Molecular Biology 1.9k
Replace Jakob Dupont with:
Jakob Dupont United States
Efisio Puxeddu Italy
Elizabeth Mambo United States
Hiroyuki Namba Japan
Éric Letouzé France
H. Rabes Germany
Mathilde Sibony France
Valentine M. Macaulay United Kingdom
Pernilla Wikström Sweden
Barry R. Davies United Kingdom
Valdemar Máximo relative to Jakob Dupont United States Jakob Dupont's profile →
Citations per field
00.5×1.7×
Jakob Dupont · 1×
Citations per year

Countries citing papers authored by Valdemar Máximo

Since Specialization
Citations

This map shows the geographic impact of Valdemar Máximo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Valdemar Máximo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Valdemar Máximo more than expected).

Fields of papers citing papers by Valdemar Máximo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Valdemar Máximo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Valdemar Máximo. The network helps show where Valdemar Máximo may publish in the future.

Co-authors

The 25 scholars most cited alongside Valdemar Máximo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Valdemar Máximo Line = papers co-authored together Valdemar Máximo links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 83 papers — load more, or switch the sort, to bring in the rest.

#Work
1
BRAF mutations and RET/PTC rearrangements are alternative events in the etiopathogenesis of PTC
Hit paper breakdown →
2003533
2 2004297
3 2002203
4 2005203
5 2015158
6 2005156
7 2004151
8 2012112
9 2009110
10 2009105
11 2000101
12 200199
13 201594
14 201491
15 202089
16 200784
17 200881
18 201869
19 201168
20 201268

About Valdemar Máximo

Valdemar Máximo is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism, Oncology, Cancer Research and Surgery, having authored 83 papers that have together received 4.1k indexed citations. Recurring topics across this work include Thyroid Cancer Diagnosis and Treatment (34 papers), Cancer-related Molecular Pathways (17 papers), Cancer, Hypoxia, and Metabolism (17 papers), Genetic factors in colorectal cancer (11 papers), Mitochondrial Function and Pathology (11 papers), RNA modifications and cancer (9 papers), BRCA gene mutations in cancer (5 papers) and ATP Synthase and ATPases Research (4 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (2.0k citations), Cancer Research (753 citations), Oncology (1.1k citations), Pathology and Forensic Medicine (512 citations) and Molecular Biology (1.9k citations). Valdemar Máximo has collaborated with scholars based in Portugal, Brazil and United Kingdom. Frequent co-authors include Manuel Sobrinho‐Simões, Paula Soares, Jorge Lima, Vítor Trovisco, Patrícia Castro, Ana Preto, Ana Sofia Rocha, Tiago O. Botelho, Raquel Seruca and José Cameselle‐Teijeiro. Their work appears in journals such as Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin, The Journal of Pathology, Mitochondrion, The Journal of Clinical Endocrinology & Metabolism and Frontiers in Endocrinology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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