Tsuneyasu Kaisho

53.0k citations
167 papers · 40.6k · 13 hit papers · h-index 73

Impact in

  • Immunology top 0.01%
    • Immune Response and Inflammation
    • Immune Cell Function and Interaction
    • Immunotherapy and Immune Responses
    • interferon and immune responses
    • T-cell and B-cell Immunology
  • Microbiology top 0.05%
    • Antimicrobial Peptides and Activities

Papers in

    • Immune Response and Inflammation 73
    • Immunotherapy and Immune Responses 64
    • Immune Cell Function and Interaction 62
    • T-cell and B-cell Immunology 43
    • interferon and immune responses 18
    • Immune cells in cancer 11
    • NF-κB Signaling Pathways 29

Tsuneyasu Kaisho

163 papers receiving 39.8k citations

Tsuneyasu Kaisho's Hit Papers

Critical Role for CD103+/CD141+ Dendritic Cells Bearing CCR7 for Tumor Antigen Trafficking and Priming of T Cell Immunity in Melanoma 2016 · 747 citations
7470+9+18Years since publication10002.0k3.0k4.0k5.0k

Peers

Tsuneyasu Kaisho
Comparison fields: 5 of 154
  • Immunology 28.6k
  • Microbiology 2.4k
  • Cancer Research 4.5k
  • Oncology 4.7k
  • Epidemiology 5.5k
Replace Shintaro Sato with:
Shintaro Sato Japan
Satoshi Uematsu Japan
Hiroaki Hemmi Japan
Bruce Beutler United States
Masahiro Yamamoto Japan
Katsuaki Hoshino Japan
Douglas T. Golenbock United States
Yoichiro Iwakura Japan
Hermann Wagner Germany
Stefanie N. Vogel United States
Tsuneyasu Kaisho relative to Shintaro Sato Japan Shintaro Sato's profile →
Citations per field
00.5×1.5×
Shintaro Sato · 1×
Citations per year

Countries citing papers authored by Tsuneyasu Kaisho

Since Specialization
Citations

This map shows the geographic impact of Tsuneyasu Kaisho's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tsuneyasu Kaisho with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tsuneyasu Kaisho more than expected).

Fields of papers citing papers by Tsuneyasu Kaisho

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tsuneyasu Kaisho. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tsuneyasu Kaisho. The network helps show where Tsuneyasu Kaisho may publish in the future.

Co-authors

The 25 scholars most cited alongside Tsuneyasu Kaisho, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tsuneyasu Kaisho Line = papers co-authored together Tsuneyasu Kaisho links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 167 papers — load more, or switch the sort, to bring in the rest.

#Work
1
A Toll-like receptor recognizes bacterial DNA
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20005268
2
Toll-Like Receptors
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20035024
3
Toll-like receptors: critical proteins linking innate and acquired immunity
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20013885
4
Innate Antiviral Responses by Means of TLR7-Mediated Recognition of Single-Stranded RNA
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20042692
5
Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling Pathway
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20032621
6
Small anti-viral compounds activate immune cells via the TLR7 MyD88–dependent signaling pathway
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20021983
7
Enhanced Th1 Activity and Development of Chronic Enterocolitis in Mice Devoid of Stat3 in Macrophages and Neutrophils
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19991012
8
TRAM is specifically involved in the Toll-like receptor 4–mediated MyD88-independent signaling pathway
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2003828
9
Essential role for TIRAP in activation of the signalling cascade shared by TLR2 and TLR4
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2002803
10
Critical Role for CD103+/CD141+ Dendritic Cells Bearing CCR7 for Tumor Antigen Trafficking and Priming of T Cell Immunity in Melanoma
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2016747
11
Toll-like receptor function and signaling
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2006678
12
Stat3 Activation Is Responsible for IL-6-Dependent T Cell Proliferation Through Preventing Apoptosis: Generation and Characterization of T Cell-Specific Stat3-Deficient Mice
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1998527
13 2004479
14
T Cell-Specific Loss of Pten Leads to Defects in Central and Peripheral Tolerance
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2001464
15 2003454
16 2001413
17 2004410
18 2002388
19 2002358
20 1986317

About Tsuneyasu Kaisho

Tsuneyasu Kaisho is a scholar working on Immunology, Cancer Research, Molecular Biology, Oncology and Immunology and Allergy, having authored 167 papers that have together received 40.6k indexed citations. Recurring topics across this work include Immune Response and Inflammation (73 papers), Immunotherapy and Immune Responses (64 papers), Immune Cell Function and Interaction (62 papers), T-cell and B-cell Immunology (43 papers), NF-κB Signaling Pathways (29 papers), interferon and immune responses (18 papers), Immune cells in cancer (11 papers) and Cytokine Signaling Pathways and Interactions (9 papers). The work is most often cited by research in Immunology (28.6k citations), Microbiology (2.4k citations), Cancer Research (4.5k citations), Oncology (4.7k citations) and Epidemiology (5.5k citations). Tsuneyasu Kaisho has collaborated with scholars based in Japan, United States and Germany. Frequent co-authors include Shizuo Akira, Kiyoshi Takeda, Hiroaki Hemmi, Katsuaki Hoshino, Osamu Takeuchi, Shintaro Sato, Hideki Sanjo, Taro Kawai, Caetano Reis e Sousa and Sandra S. Diebold. Their work appears in journals such as The Journal of Immunology, Immunity, The Journal of Experimental Medicine, International Immunology and Frontiers in Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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