Timothy R. Billiar
Impact in
-
- Trauma, Hemostasis, Coagulopathy, Resuscitation
- Biochemistry top 0.01%
- Eicosanoids and Hypertension Pharmacology
Papers in
- Immunology 242
- Immune Response and Inflammation 165
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 45
-
- Heme Oxygenase-1 and Carbon Monoxide 53
- Co-authors
- Richard L. Simmons (64 shared papers)David A. Geller (53 shared papers)Yoram Vodovotz (130 shared papers)Allan Tsung (40 shared papers)Andreas K. Nüssler (20 shared papers)Young‐Myeong Kim (20 shared papers)Andrew B. Peitzman (98 shared papers)Ronald Curran (26 shared papers)
- Journals
- Shock (60 papers)The Journal of Trauma: Injury, Infection, and Critical Care (36 papers)Molecular Medicine (26 papers)The Journal of Immunology (26 papers)Annals of Surgery (24 papers)
- Partner nations
- United StatesChinaGermany
In The Last Decade
Timothy R. Billiar
789 papers receiving 58.7k citations
Timothy R. Billiar's Hit Papers
Peers
Comparison fields: 5 of 184
- Critical Care and Intensive Care Medicine 3.1k
- Biochemistry 5.0k
- Clinical Biochemistry 4.3k
- Immunology 12.9k
- Physiology 12.1k
Countries citing papers authored by Timothy R. Billiar
This map shows the geographic impact of Timothy R. Billiar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Timothy R. Billiar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Timothy R. Billiar more than expected).
Fields of papers citing papers by Timothy R. Billiar
This network shows the impact of papers produced by Timothy R. Billiar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Timothy R. Billiar. The network helps show where Timothy R. Billiar may publish in the future.
Co-authors
The 25 scholars most cited alongside Timothy R. Billiar, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 808 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion Hit paper breakdown → | 2005 | 972 |
| 2 | Nitric Oxide Inhibits Apoptosis by Preventing Increases in Caspase-3-like Activity via Two Distinct Mechanisms Hit paper breakdown → | 1997 | 816 |
| 3 | HMGB1 in health and disease Hit paper breakdown → | 2014 | 809 |
| 4 | Inflammation, immunoregulation, and inducible nitric oxide synthase Hit paper breakdown → | 1993 | 785 |
| 5 | The Tumor Suppressor p53 Limits Ferroptosis by Blocking DPP4 Activity Hit paper breakdown → | 2017 | 777 |
| 6 | Molecular cloning and expression of inducible nitric oxide synthase from human hepatocytes. Hit paper breakdown → | 1993 | 755 |
| 7 | Apoptosis induced in normal human hepatocytes by tumor necrosis factor-related apoptosis-inducing ligand Hit paper breakdown → | 2000 | 735 |
| 8 | HMGB1: Endogenous Danger Signaling Hit paper breakdown → | 2008 | 672 |
| 9 | Cytokines, endotoxin, and glucocorticoids regulate the expression of inducible nitric oxide synthase in hepatocytes. Hit paper breakdown → | 1993 | 611 |
| 10 | HMGB1 release induced by liver ischemia involves Toll-like receptor 4–dependent reactive oxygen species production and calcium-mediated signaling Hit paper breakdown → | 2007 | 535 |
| 11 | The grateful dead: damage‐associated molecular pattern molecules and reduction/oxidation regulate immunity Hit paper breakdown → | 2007 | 532 |
| 12 | Nitrogen Oxide Levels in Patients After Trauma and During Sepsis Hit paper breakdown → | 1991 | 532 |
| 13 | Lipid Peroxidation Drives Gasdermin D-Mediated Pyroptosis in Lethal Polymicrobial Sepsis Hit paper breakdown → | 2018 | 505 |
| 14 | 1994 | 494 | |
| 15 | 2010 | 488 | |
| 16 | 1997 | 487 | |
| 17 | 2001 | 467 | |
| 18 | 2003 | 458 | |
| 19 | 2000 | 455 | |
| 20 | PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation Hit paper breakdown → | 2016 | 449 |
About Timothy R. Billiar
Timothy R. Billiar is a scholar working on Immunology, Molecular Biology, Physiology, Epidemiology and Biochemistry, having authored 808 papers that have together received 60.3k indexed citations. Recurring topics across this work include Immune Response and Inflammation (165 papers), Nitric Oxide and Endothelin Effects (163 papers), Eicosanoids and Hypertension Pharmacology (91 papers), Advanced Glycation End Products research (66 papers), Sepsis Diagnosis and Treatment (62 papers), Heme Oxygenase-1 and Carbon Monoxide (53 papers), Trauma and Emergency Care Studies (49 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (45 papers). The work is most often cited by research in Critical Care and Intensive Care Medicine (3.1k citations), Biochemistry (5.0k citations), Clinical Biochemistry (4.3k citations), Immunology (12.9k citations) and Physiology (12.1k citations). Timothy R. Billiar has collaborated with scholars based in United States, China and Germany. Frequent co-authors include Richard L. Simmons, David A. Geller, Yoram Vodovotz, Allan Tsung, Andreas K. Nüssler, Young‐Myeong Kim, Andrew B. Peitzman, Ronald Curran, Michael T. Lotze and Brian G. Harbrecht. Their work appears in journals such as Shock, The Journal of Trauma: Injury, Infection, and Critical Care, Molecular Medicine, The Journal of Immunology and Annals of Surgery.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.