Thomas Seitz

1.3k citations
7 papers · 1.1k · 1 hit paper · h-index 7

Impact in

    • Melanoma and MAPK Pathways
    • Protein Kinase Regulation and GTPase Signaling
    • Cell death mechanisms and regulation
    • Mechanisms of cancer metastasis
    • 14-3-3 protein interactions
    • Cancer Mechanisms and Therapy

Papers in

    • Protein Kinase Regulation and GTPase Signaling 3
    • Melanoma and MAPK Pathways 2
    • Ubiquitin and proteasome pathways 1
    • 14-3-3 protein interactions 1
    • Bacterial Genetics and Biotechnology 2

Thomas Seitz

7 papers receiving 1.1k citations

Thomas Seitz's Hit Papers

Suppression of Raf-1 kinase activity and MAP kinase signalling by RKIP 1999 · 746 citations
7460+9+18Years since publication200400600

Peers

Thomas Seitz
Comparison fields: 5 of 89
  • Molecular Biology 841
  • Pathology and Forensic Medicine 195
  • Cancer Research 105
  • Cell Biology 99
  • Oncology 141
Replace Kostas D. Katsanakis with:
Kostas D. Katsanakis United Kingdom
Frances Fee United Kingdom
Paolo Bonvini Italy
Gautam Sondarva United States
Michelle Wong United States
Christopher Leo United States
Iris Eisenmann-Tappe Germany
Eldon R. Jupe United States
Christian Kaiser Germany
Yumiko Hirokawa Australia
Thomas Seitz relative to Kostas D. Katsanakis United Kingdom Kostas D. Katsanakis's profile →
Citations per field
00.5×1.5×
Kostas D. Katsanakis · 1×
Citations per year

Countries citing papers authored by Thomas Seitz

Since Specialization
Citations

This map shows the geographic impact of Thomas Seitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Seitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Seitz more than expected).

Fields of papers citing papers by Thomas Seitz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Seitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Seitz. The network helps show where Thomas Seitz may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas Seitz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas Seitz Line = papers co-authored together Thomas Seitz links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1
Suppression of Raf-1 kinase activity and MAP kinase signalling by RKIP
Hit paper breakdown →
1999746
2 1996186
3 199666
4 199639
5 199821
6 202110
7 20007

About Thomas Seitz

Thomas Seitz is a scholar working on Molecular Biology, Genetics, Pathology and Forensic Medicine, Ecology and Biochemistry, having authored 7 papers that have together received 1.1k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (3 papers), Melanoma and MAPK Pathways (2 papers), Bacterial Genetics and Biotechnology (2 papers), Cancer Mechanisms and Therapy (1 paper), Ubiquitin and proteasome pathways (1 paper), NF-κB Signaling Pathways (1 paper), Amino Acid Enzymes and Metabolism (1 paper) and 14-3-3 protein interactions (1 paper). The work is most often cited by research in Molecular Biology (841 citations), Pathology and Forensic Medicine (195 citations), Cancer Research (105 citations), Cell Biology (99 citations) and Oncology (141 citations). Thomas Seitz has collaborated with scholars based in Switzerland, Germany and United States. Frequent co-authors include Harald Mischak, Walter Kölch, Petra Janosch, John M. Sedivy, David W. Rose, Christian Kaiser, Shengfeng Li, Kostas D. Katsanakis, Kam C. Yeung and Brian W. McFerran. Their work appears in journals such as Molecular and Cellular Biology, Protein Engineering Design and Selection, Journal of Biological Chemistry, Journal of Molecular Biology and Nature.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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