Thomas Østerbye
Impact in
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- Immune Cell Function and Interaction
- T-cell and B-cell Immunology
- Immunotherapy and Immune Responses
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- Diabetes and associated disorders
Papers in
- Co-authors
- Maria Blomqvist (5 shared papers)Karsten Buschard (8 shared papers)Pam Fredman (5 shared papers)Søren Buus (11 shared papers)Manfred Brigl (1 shared paper)Jan‐Eric Månsson (4 shared papers)Susanna Cardell (1 shared paper)Sara Rhost (1 shared paper)
In The Last Decade
Thomas Østerbye
20 papers receiving 369 citations
Peers
Comparison fields: 5 of 56
- Immunology 177
- Genetics 94
- Endocrinology, Diabetes and Metabolism 50
- Gastroenterology 12
- Surgery 94
Countries citing papers authored by Thomas Østerbye
This map shows the geographic impact of Thomas Østerbye's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Østerbye with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Østerbye more than expected).
Fields of papers citing papers by Thomas Østerbye
This network shows the impact of papers produced by Thomas Østerbye. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Østerbye. The network helps show where Thomas Østerbye may publish in the future.
Co-authors
The 25 scholars most cited alongside Thomas Østerbye, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 21 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 87 | |
| 2 | 2001 | 41 | |
| 3 | 2005 | 41 | |
| 4 | 2003 | 23 | |
| 5 | 2010 | 22 | |
| 6 | 2015 | 20 | |
| 7 | 2020 | 19 | |
| 8 | 2012 | 19 | |
| 9 | 2011 | 15 | |
| 10 | 2002 | 13 | |
| 11 | 2013 | 12 | |
| 12 | 2020 | 11 | |
| 13 | 2017 | 9 | |
| 14 | 2016 | 9 | |
| 15 | 2006 | 8 | |
| 16 | 2022 | 8 | |
| 17 | 2020 | 7 | |
| 18 | 2011 | 5 | |
| 19 | 2023 | 3 | |
| 20 | 2015 | 2 |
About Thomas Østerbye
Thomas Østerbye is a scholar working on Genetics, Surgery, Molecular Biology, Immunology and Radiology, Nuclear Medicine and Imaging, having authored 21 papers that have together received 374 indexed citations. Recurring topics across this work include Diabetes and associated disorders (9 papers), Pancreatic function and diabetes (8 papers), Immune Cell Function and Interaction (7 papers), Monoclonal and Polyclonal Antibodies Research (5 papers), vaccines and immunoinformatics approaches (4 papers), T-cell and B-cell Immunology (4 papers), Diet, Metabolism, and Disease (3 papers) and Advanced Biosensing Techniques and Applications (3 papers). The work is most often cited by research in Immunology (177 citations), Genetics (94 citations), Endocrinology, Diabetes and Metabolism (50 citations), Gastroenterology (12 citations) and Surgery (94 citations). Thomas Østerbye has collaborated with scholars based in Denmark, Sweden and Argentina. Frequent co-authors include Maria Blomqvist, Karsten Buschard, Pam Fredman, Søren Buus, Manfred Brigl, Jan‐Eric Månsson, Susanna Cardell, Sara Rhost, Jan‐Eric Månsson and Susann Teneberg. Their work appears in journals such as PLoS ONE, Frontiers in Immunology, Diabetes, Lipids in Health and Disease and Diabetes/Metabolism Research and Reviews.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.