Thomas E. Callis

6.9k citations
27 papers · 5.8k · 2 hit papers · h-index 22

Impact in

Papers in

Thomas E. Callis

27 papers receiving 5.6k citations

Thomas E. Callis's Hit Papers

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice 2009 · 738 citations
7380+7+14Years since publication5001k1.5k2k

Peers

Thomas E. Callis
Comparison fields: 5 of 100
  • Cancer Research 2.3k
  • Cardiology and Cardiovascular Medicine 1.3k
  • Molecular Biology 2.4k
  • Genetics 365
  • Rehabilitation 66
Replace Lillian B. Sutherland with:
Lillian B. Sutherland United States
Jan Krützfeldt Switzerland
Simona Greco Italy
Elizabeth M. Mandel United States
Danish H Sayed United States
Shizuka Uchida Denmark
Yasushi Matsuki Japan
Venus Ka-Man Lai United States
Djamel Lebeche United States
Sheng‐Da Hsu Taiwan
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Citations per field
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Citations per year

Countries citing papers authored by Thomas E. Callis

Since Specialization
Citations

This map shows the geographic impact of Thomas E. Callis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas E. Callis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas E. Callis more than expected).

Fields of papers citing papers by Thomas E. Callis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas E. Callis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas E. Callis. The network helps show where Thomas E. Callis may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas E. Callis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas E. Callis Line = papers co-authored together Thomas E. Callis links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 27 papers — load more, or switch the sort, to bring in the rest.

#Work
1
The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation
Hit paper breakdown →
20052371
2
MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice
Hit paper breakdown →
2009738
3 2008487
4 2007378
5 2009311
6 2011250
7 2007142
8 2016141
9 2008127
10 2008114
11 2008102
12 200895
13 201586
14 201183
15 202258
16 201953
17 200545
18 201045
19 201444
20 201839

About Thomas E. Callis

Thomas E. Callis is a scholar working on Cardiology and Cardiovascular Medicine, Cancer Research, Molecular Biology, Genetics and Surgery, having authored 27 papers that have together received 5.8k indexed citations. Recurring topics across this work include Cardiac electrophysiology and arrhythmias (7 papers), Muscle Physiology and Disorders (6 papers), MicroRNA in disease regulation (5 papers), Cardiomyopathy and Myosin Studies (5 papers), Genomics and Rare Diseases (4 papers), Congenital heart defects research (4 papers), Cardiac Fibrosis and Remodeling (3 papers) and Signaling Pathways in Disease (1 paper). The work is most often cited by research in Cancer Research (2.3k citations), Cardiology and Cardiovascular Medicine (1.3k citations), Molecular Biology (2.4k citations), Genetics (365 citations) and Rehabilitation (66 citations). Thomas E. Callis has collaborated with scholars based in United States, Netherlands and China. Frequent co-authors include Jian‐Fu Chen, Scott M. Hammond, J. Michael Thomson, Elizabeth M. Mandel, Frank L. Conlon, Da-Zhi Wang, Mariko Tatsuguchi, Zhong‐Liang Deng, Heeyoung Seok and Ruhang Tang. Their work appears in journals such as Circulation Genomic and Precision Medicine, JAMA Cardiology, Journal of Cardiovascular Translational Research, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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