Thomas Conrad
Impact in
- Cancer Research top 10%
- Cancer-related molecular mechanisms research
- Molecular Biology top 10%
- RNA modifications and cancer
- RNA Research and Splicing
- Genomics and Chromatin Dynamics
- RNA and protein synthesis mechanisms
- Epigenetics and DNA Methylation
Papers in
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- RNA Research and Splicing 7
- Single-cell and spatial transcriptomics 5
- Ubiquitin and proteasome pathways 4
- Genomics and Chromatin Dynamics 4
- RNA modifications and cancer 4
- RNA and protein synthesis mechanisms 3
-
- Cancer-related molecular mechanisms research 4
- Co-authors
- Asifa Akhtar (6 shared papers)Ulf Andersson Ørom (7 shared papers)Evgenia Ntini (3 shared papers)Annita Louloupi (3 shared papers)Sascha Sauer (4 shared papers)Nicholas M. Luscombe (4 shared papers)Juan M. Vaquerizas (3 shared papers)Ramón Vidal (3 shared papers)
- Journals
- Nature Communications (4 papers)Cell Reports (3 papers)Journal of Neuroinflammation (2 papers)Journal of Clinical Investigation (2 papers)RNA (2 papers)
- Partner nations
- GermanyUnited StatesAustria
In The Last Decade
Thomas Conrad
32 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 83
- Cancer Research 251
- Molecular Biology 1.1k
- Aging 22
- Genetics 193
- Neurology 44
Countries citing papers authored by Thomas Conrad
This map shows the geographic impact of Thomas Conrad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Conrad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Conrad more than expected).
Fields of papers citing papers by Thomas Conrad
This network shows the impact of papers produced by Thomas Conrad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Conrad. The network helps show where Thomas Conrad may publish in the future.
Co-authors
The 25 scholars most cited alongside Thomas Conrad, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 204 | |
| 2 | 2018 | 174 | |
| 3 | 2019 | 117 | |
| 4 | 2010 | 117 | |
| 5 | 2016 | 105 | |
| 6 | 2022 | 78 | |
| 7 | 2014 | 67 | |
| 8 | 2012 | 61 | |
| 9 | 2020 | 59 | |
| 10 | 2016 | 58 | |
| 11 | 2008 | 57 | |
| 12 | 2012 | 55 | |
| 13 | 2014 | 50 | |
| 14 | 2023 | 34 | |
| 15 | 2023 | 30 | |
| 16 | 2016 | 18 | |
| 17 | 2022 | 17 | |
| 18 | 2022 | 12 | |
| 19 | 2024 | 12 | |
| 20 | 2022 | 12 |
About Thomas Conrad
Thomas Conrad is a scholar working on Molecular Biology, Cancer Research, Oncology, Neurology and Immunology, having authored 36 papers that have together received 1.4k indexed citations. Recurring topics across this work include RNA Research and Splicing (7 papers), Single-cell and spatial transcriptomics (5 papers), Neuroinflammation and Neurodegeneration Mechanisms (5 papers), Ubiquitin and proteasome pathways (4 papers), Cancer-related molecular mechanisms research (4 papers), Genomics and Chromatin Dynamics (4 papers), RNA modifications and cancer (4 papers) and RNA and protein synthesis mechanisms (3 papers). The work is most often cited by research in Cancer Research (251 citations), Molecular Biology (1.1k citations), Aging (22 citations), Genetics (193 citations) and Neurology (44 citations). Thomas Conrad has collaborated with scholars based in Germany, United States and Austria. Frequent co-authors include Asifa Akhtar, Ulf Andersson Ørom, Evgenia Ntini, Annita Louloupi, Sascha Sauer, Nicholas M. Luscombe, Juan M. Vaquerizas, Ramón Vidal, Herbert Holz and David Meierhofer. Their work appears in journals such as Nature Communications, Cell Reports, Journal of Neuroinflammation, Journal of Clinical Investigation and RNA.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.