Ted Petley

521 citations
8 papers · 409 · h-index 7

Impact in

Papers in

    • IL-33, ST2, and ILC Pathways 2
    • Galectins and Cancer Biology 1
    • Psoriasis: Treatment and Pathogenesis 1
    • Protein Kinase Regulation and GTPase Signaling 2
    • FOXO transcription factor regulation 1
    • Muscle Physiology and Disorders 1

Ted Petley

8 papers receiving 402 citations

Peers

Ted Petley
Comparison fields: 5 of 58
  • Physiology 173
  • Immunology and Allergy 38
  • Immunology 134
  • Endocrinology, Diabetes and Metabolism 75
  • Dermatology 21
Replace Bernd Schnarr with:
Bernd Schnarr Germany
F. Paolieri Italy
Justine Van Moorleghem Belgium
Osamu Taniguchi Japan
Andrea Mogas Canada
Benjamin Schrijver Netherlands
Paul T.M. Kok Netherlands
Andrea G. Bree United States
Barbara Iwasiow Canada
Ana P. Lopes Netherlands
Ted Petley relative to Bernd Schnarr Germany Bernd Schnarr's profile →
Citations per field
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Citations per year

Countries citing papers authored by Ted Petley

Since Specialization
Citations

This map shows the geographic impact of Ted Petley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ted Petley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ted Petley more than expected).

Fields of papers citing papers by Ted Petley

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ted Petley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ted Petley. The network helps show where Ted Petley may publish in the future.

Co-authors

The 25 scholars most cited alongside Ted Petley, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ted Petley Line = papers co-authored together Ted Petley links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 2004163
2 1999102
3 200574
4 199931
5 200816
6 201016
7 20166
8 20241

About Ted Petley

Ted Petley is a scholar working on Immunology, Molecular Biology, Physiology, Immunology and Allergy and Surgery, having authored 8 papers that have together received 409 indexed citations. Recurring topics across this work include Asthma and respiratory diseases (3 papers), Allergic Rhinitis and Sensitization (2 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), IL-33, ST2, and ILC Pathways (2 papers), Galectins and Cancer Biology (1 paper), FOXO transcription factor regulation (1 paper), Muscle Physiology and Disorders (1 paper) and Psoriasis: Treatment and Pathogenesis (1 paper). The work is most often cited by research in Physiology (173 citations), Immunology and Allergy (38 citations), Immunology (134 citations), Endocrinology, Diabetes and Metabolism (75 citations) and Dermatology (21 citations). Ted Petley has collaborated with scholars based in United States, Canada and France. Frequent co-authors include James R. Florini, Jill Giles‐Komar, Gaoyun Yang, A. Paige Davis Volk, Anuk Das, Lily Li, Don E. Griswold, Eva Emmell, Xiaozhou Shang and Li J. Their work appears in journals such as Hormone and Metabolic Research, Molecular Immunology, European Journal of Cancer, Cytokine and Cellular Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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