Tamara Leclercq

717 citations
14 papers · 564 · h-index 8

Impact in

    • Endoplasmic Reticulum Stress and Disease
    • Cellular transport and secretion
    • Sphingolipid Metabolism and Signaling
    • Lipid Membrane Structure and Behavior
    • Protein Kinase Regulation and GTPase Signaling

Papers in

    • Chronic Myeloid Leukemia Treatments 8
    • Acute Myeloid Leukemia Research 2
    • Sphingolipid Metabolism and Signaling 6
    • Lipid Membrane Structure and Behavior 5
    • Protein Kinase Regulation and GTPase Signaling 2

Tamara Leclercq

14 papers receiving 560 citations

Peers

Tamara Leclercq
Comparison fields: 5 of 63
  • Cell Biology 249
  • Molecular Biology 487
  • Hematology 50
  • Physiology 17
  • Immunology 51
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Citations per field
00.5×5.7×
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Citations per year

Countries citing papers authored by Tamara Leclercq

Since Specialization
Citations

This map shows the geographic impact of Tamara Leclercq's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tamara Leclercq with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tamara Leclercq more than expected).

Fields of papers citing papers by Tamara Leclercq

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tamara Leclercq. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tamara Leclercq. The network helps show where Tamara Leclercq may publish in the future.

Co-authors

The 25 scholars most cited alongside Tamara Leclercq, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tamara Leclercq Line = papers co-authored together Tamara Leclercq links everyone, so they are left out of the graph.

All Works

14 of 14 papers shown
#Work
1 2004230
2 2010103
3 201651
4 200649
5 200845
6 201234
7 201925
8 201711
9 20176
10 20213
11 20163
12 20152
13 20151
14 20141

About Tamara Leclercq

Tamara Leclercq is a scholar working on Hematology, Molecular Biology, Genetics, Cell Biology and Organic Chemistry, having authored 14 papers that have together received 564 indexed citations. Recurring topics across this work include Chronic Myeloid Leukemia Treatments (8 papers), Sphingolipid Metabolism and Signaling (6 papers), Lipid Membrane Structure and Behavior (5 papers), Cellular transport and secretion (4 papers), Chronic Lymphocytic Leukemia Research (4 papers), Acute Myeloid Leukemia Research (2 papers), Acute Lymphoblastic Leukemia research (2 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). The work is most often cited by research in Cell Biology (249 citations), Molecular Biology (487 citations), Hematology (50 citations), Physiology (17 citations) and Immunology (51 citations). Tamara Leclercq has collaborated with scholars based in Australia, United States and United Kingdom. Frequent co-authors include Stuart M. Pitson, Paul A.B. Moretti, Mathew A. Vadas, Julia R. Zebol, Binks W. Wattenberg, Pu Xia, Francesca Tonelli, Keng Gat Lim, Susan Pyne and Nigel J. Pyne. Their work appears in journals such as Blood, Journal of Biological Chemistry, The Journal of Experimental Medicine, Blood Advances and IUBMB Life.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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