Tamara Hansmann
Impact in
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- Genetic Syndromes and Imprinting
- BRCA gene mutations in cancer
- Animal Genetics and Reproduction
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- Epigenetics and DNA Methylation
Papers in
- Genetics 5
- Genetic Syndromes and Imprinting 4
- BRCA gene mutations in cancer 1
- Animal Genetics and Reproduction 1
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- Epigenetics and DNA Methylation 6
- Co-authors
- Thomas Haaf (9 shared papers)Ulrich Zechner (5 shared papers)J. Heinzmann (5 shared papers)C. Wrenzycki (3 shared papers)Hendrik J. Niemann (2 shared papers)Galyna Pliushch (1 shared paper)Sabine Preisler-Adams (1 shared paper)Andrea Gehrig (1 shared paper)
In The Last Decade
Tamara Hansmann
10 papers receiving 343 citations
Peers
Comparison fields: 5 of 34
- Genetics 120
- Molecular Biology 151
- Public Health, Environmental and Occupational Health 71
- Agronomy and Crop Science 18
- Pediatrics, Perinatology and Child Health 17
Countries citing papers authored by Tamara Hansmann
This map shows the geographic impact of Tamara Hansmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tamara Hansmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tamara Hansmann more than expected).
Fields of papers citing papers by Tamara Hansmann
This network shows the impact of papers produced by Tamara Hansmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tamara Hansmann. The network helps show where Tamara Hansmann may publish in the future.
Co-authors
The 25 scholars most cited alongside Tamara Hansmann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 90 | |
| 2 | 2011 | 64 | |
| 3 | 2011 | 49 | |
| 4 | 2012 | 41 | |
| 5 | 2008 | 33 | |
| 6 | 2012 | 30 | |
| 7 | 2009 | 22 | |
| 8 | 2010 | 14 | |
| 9 | 2013 | 3 | |
| 10 | 2009 | 1 |
About Tamara Hansmann
Tamara Hansmann is a scholar working on Genetics, Molecular Biology, Public Health, Environmental and Occupational Health, Hematology and Pharmacology, having authored 10 papers that have together received 347 indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (6 papers), Genetic Syndromes and Imprinting (4 papers), Reproductive Biology and Fertility (2 papers), BRCA gene mutations in cancer (1 paper), Hemophilia Treatment and Research (1 paper), Animal Genetics and Reproduction (1 paper), Electrochemical sensors and biosensors (1 paper) and Cholinesterase and Neurodegenerative Diseases (1 paper). The work is most often cited by research in Genetics (120 citations), Molecular Biology (151 citations), Public Health, Environmental and Occupational Health (71 citations), Agronomy and Crop Science (18 citations) and Pediatrics, Perinatology and Child Health (17 citations). Tamara Hansmann has collaborated with scholars based in Germany, France and Slovenia. Frequent co-authors include Thomas Haaf, Ulrich Zechner, J. Heinzmann, C. Wrenzycki, Hendrik J. Niemann, Galyna Pliushch, Sabine Preisler-Adams, Andrea Gehrig, Daniela Endt and Peter F. Wieacker. Their work appears in journals such as Epigenetics, Cytogenetic and Genome Research, Reproduction, Reproduction Fertility and Development and Biosensors and Bioelectronics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.