Simon Low
Impact in
- Cell Biology top 10%
- Microtubule and mitosis dynamics
-
- Phosphodiesterase function and regulation
- Ubiquitin and proteasome pathways
- Protein Degradation and Inhibitors
Papers in
-
- Ion channel regulation and function 1
- Melanoma and MAPK Pathways 1
- Oncology 3
- Cancer-related Molecular Pathways 2
- Peptidase Inhibition and Analysis 1
- Cancer Treatment and Pharmacology 1
- Co-authors
- Yuanhua Ding (4 shared papers)Michael Kothe (3 shared papers)Darcy Kohls (3 shared papers)Cyrille Kuhn (3 shared papers)Rocco Coli (3 shared papers)Glen R. Rennie (2 shared papers)Frédéric Féru (2 shared papers)Mian Gao (2 shared papers)
- Journals
- Biochemistry (2 papers)Molecular Pharmacology (1 paper)Journal of Pharmacology and Experimental Therapeutics (1 paper)Bioorganic & Medicinal Chemistry Letters (1 paper)Chemical Biology & Drug Design (1 paper)
- Partner nations
- United States
In The Last Decade
Simon Low
6 papers receiving 363 citations
Peers
Comparison fields: 5 of 49
- Cell Biology 116
- Molecular Biology 239
- Oncology 86
- Computational Theory and Mathematics 45
- Surgery 82
Countries citing papers authored by Simon Low
This map shows the geographic impact of Simon Low's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simon Low with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simon Low more than expected).
Fields of papers citing papers by Simon Low
This network shows the impact of papers produced by Simon Low. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simon Low. The network helps show where Simon Low may publish in the future.
Co-authors
The 25 scholars most cited alongside Simon Low, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2007 | 104 | |
| 2 | 2014 | 103 | |
| 3 | 2007 | 95 | |
| 4 | 2005 | 46 | |
| 5 | 2017 | 11 | |
| 6 | 2010 | 9 |
About Simon Low
Simon Low is a scholar working on Molecular Biology, Oncology, Cell Biology, Surgery and Pathology and Forensic Medicine, having authored 6 papers that have together received 368 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (2 papers), Microtubule and mitosis dynamics (2 papers), Peptidase Inhibition and Analysis (1 paper), Ion channel regulation and function (1 paper), Cancer Treatment and Pharmacology (1 paper), Neuroscience and Neural Engineering (1 paper), Computational Drug Discovery Methods (1 paper) and Melanoma and MAPK Pathways (1 paper). The work is most often cited by research in Cell Biology (116 citations), Molecular Biology (239 citations), Oncology (86 citations), Computational Theory and Mathematics (45 citations) and Surgery (82 citations). Simon Low has collaborated with scholars based in United States. Frequent co-authors include Yuanhua Ding, Michael Kothe, Darcy Kohls, Cyrille Kuhn, Rocco Coli, Glen R. Rennie, Frédéric Féru, Mian Gao, Thomas A. Wynn and Christine Loh. Their work appears in journals such as Biochemistry, Molecular Pharmacology, Journal of Pharmacology and Experimental Therapeutics, Bioorganic & Medicinal Chemistry Letters and Chemical Biology & Drug Design.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.