Henrik Moebitz

618 citations
8 papers · 441 · h-index 5

Impact in

    • Melanoma and MAPK Pathways
    • Protein Kinase Regulation and GTPase Signaling
    • PI3K/AKT/mTOR signaling in cancer
    • Protein Degradation and Inhibitors

Papers in

    • Melanoma and MAPK Pathways 4
    • PI3K/AKT/mTOR signaling in cancer 2
    • Protein Degradation and Inhibitors 1
    • Cytokine Signaling Pathways and Interactions 2
    • Cancer-related Molecular Pathways 1

Henrik Moebitz

7 papers receiving 418 citations

Peers

Henrik Moebitz
Comparison fields: 5 of 61
  • Molecular Biology 325
  • Oncology 119
  • Computational Theory and Mathematics 58
  • Organic Chemistry 105
  • Pathology and Forensic Medicine 54
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Citations per field
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Citations per year

Countries citing papers authored by Henrik Moebitz

Since Specialization
Citations

This map shows the geographic impact of Henrik Moebitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Henrik Moebitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Henrik Moebitz more than expected).

Fields of papers citing papers by Henrik Moebitz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Henrik Moebitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Henrik Moebitz. The network helps show where Henrik Moebitz may publish in the future.

Co-authors

The 25 scholars most cited alongside Henrik Moebitz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Henrik Moebitz Line = papers co-authored together Henrik Moebitz links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 2015265
2 201887
3 200835
4 200935
5 201214
6 20154
7 20111
8 20250

About Henrik Moebitz

Henrik Moebitz is a scholar working on Molecular Biology, Oncology, Organic Chemistry, Radiology, Nuclear Medicine and Imaging and Pathology and Forensic Medicine, having authored 8 papers that have together received 441 indexed citations. Recurring topics across this work include Melanoma and MAPK Pathways (4 papers), Cytokine Signaling Pathways and Interactions (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), PI3K/AKT/mTOR signaling in cancer (2 papers), Protein Degradation and Inhibitors (1 paper), Computational Drug Discovery Methods (1 paper), Cancer-related Molecular Pathways (1 paper) and Click Chemistry and Applications (1 paper). The work is most often cited by research in Molecular Biology (325 citations), Oncology (119 citations), Computational Theory and Mathematics (58 citations), Organic Chemistry (105 citations) and Pathology and Forensic Medicine (54 citations). Henrik Moebitz has collaborated with scholars based in Switzerland, United States and Norway. Frequent co-authors include Doriano Fabbro, Sandra W. Cowan‐Jacob, Láśzló Révész, Roland Feifel, Achim Schlapbach, Guido Koch, Stuart Hawtin, Clemens Scheufler, Juraj Velcicky and Christine Huppertz. Their work appears in journals such as Bioorganic & Medicinal Chemistry Letters, European Journal of Medicinal Chemistry, British Journal of Pharmacology, Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics and IUCrJ.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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