Sara E. Dobbins

6.1k citations
22 papers · 996 · h-index 16

Impact in

Papers in

    • Genomic variations and chromosomal abnormalities 3
    • Genomics and Rare Diseases 3
    • Wnt/β-catenin signaling in development and cancer 2
    • Genomics and Chromatin Dynamics 2

Sara E. Dobbins

22 papers receiving 982 citations

Peers

Sara E. Dobbins
Comparison fields: 5 of 94
  • Pathology and Forensic Medicine 216
  • Hematology 119
  • Cancer Research 153
  • Spectroscopy 126
  • Molecular Biology 463
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Citations per field
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Citations per year

Countries citing papers authored by Sara E. Dobbins

Since Specialization
Citations

This map shows the geographic impact of Sara E. Dobbins's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara E. Dobbins with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara E. Dobbins more than expected).

Fields of papers citing papers by Sara E. Dobbins

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara E. Dobbins. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara E. Dobbins. The network helps show where Sara E. Dobbins may publish in the future.

Co-authors

The 25 scholars most cited alongside Sara E. Dobbins, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Sara E. Dobbins Line = papers co-authored together Sara E. Dobbins links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 22 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2008182
2 2016126
3 2005123
4 201574
5 201864
6 201663
7 201843
8 201341
9 201940
10 201239
11 201134
12 201333
13 202223
14 201421
15 201020
16 201118
17 201914
18 201012
19 201610
20 20179

About Sara E. Dobbins

Sara E. Dobbins is a scholar working on Genetics, Molecular Biology, Pathology and Forensic Medicine, Cancer Research and Public Health, Environmental and Occupational Health, having authored 22 papers that have together received 996 indexed citations. Recurring topics across this work include Cancer Genomics and Diagnostics (7 papers), Genetic factors in colorectal cancer (6 papers), Genomic variations and chromosomal abnormalities (3 papers), Genomics and Rare Diseases (3 papers), Acute Lymphoblastic Leukemia research (3 papers), Multiple Myeloma Research and Treatments (3 papers), Wnt/β-catenin signaling in development and cancer (2 papers) and Genomics and Chromatin Dynamics (2 papers). The work is most often cited by research in Pathology and Forensic Medicine (216 citations), Hematology (119 citations), Cancer Research (153 citations), Spectroscopy (126 citations) and Molecular Biology (463 citations). Sara E. Dobbins has collaborated with scholars based in United Kingdom, United States and Canada. Frequent co-authors include Richard S. Houlston, Michael J.E. Sternberg, Daniel Chubb, Peter Broderick, Ben Kinnersley, Amy L. Sherborne, Ian Tomlinson, Alex J. Cornish, Francesco Mauri and Chris J. Pickard. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Communications, Journal of Clinical Oncology, Blood Cancer Journal and British Medical Bulletin.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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