Samuel Quentin
Impact in
- Hematology top 5%
- Acute Myeloid Leukemia Research
- Genetics top 10%
- Myeloproliferative Neoplasms: Diagnosis and Treatment
Papers in
-
- Acute Myeloid Leukemia Research 6
- Chronic Myeloid Leukemia Treatments 1
- Genetics 4
- Myeloproliferative Neoplasms: Diagnosis and Treatment 3
- Blood disorders and treatments 1
- Co-authors
- Pierre Fenaux (5 shared papers)Hervé Dombret (4 shared papers)Nicolas Boissel (3 shared papers)Jean Soulier (6 shared papers)Anna Raimbault (2 shared papers)Raphaël Itzykson (3 shared papers)Marie Passet (2 shared papers)Lionel Adès (3 shared papers)
- Journals
- Blood (4 papers)Leukemia (1 paper)Leukemia Research (1 paper)European Journal of Human Genetics (1 paper)Nature Communications (1 paper)
- Partner nations
- FranceAustraliaNetherlands
In The Last Decade
Samuel Quentin
9 papers receiving 287 citations
Peers
Comparison fields: 5 of 34
- Hematology 182
- Genetics 87
- Cancer Research 34
- Molecular Biology 132
- Oncology 39
Countries citing papers authored by Samuel Quentin
This map shows the geographic impact of Samuel Quentin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel Quentin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel Quentin more than expected).
Fields of papers citing papers by Samuel Quentin
This network shows the impact of papers produced by Samuel Quentin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel Quentin. The network helps show where Samuel Quentin may publish in the future.
Co-authors
The 25 scholars most cited alongside Samuel Quentin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 123 | |
| 2 | 2010 | 98 | |
| 3 | 2011 | 36 | |
| 4 | The Cl^-/HCO_3^- exchanger pendrin in the rat kidney is regulated in response to chronic alterations in chloride balance | 2004 | 9 |
| 5 | 2022 | 8 | |
| 6 | 2009 | 6 | |
| 7 | 2018 | 6 | |
| 8 | 2018 | 1 | |
| 9 | 2011 | 1 |
About Samuel Quentin
Samuel Quentin is a scholar working on Hematology, Genetics, Molecular Biology, Infectious Diseases and Surgery, having authored 9 papers that have together received 288 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (6 papers), Myeloproliferative Neoplasms: Diagnosis and Treatment (3 papers), Lymphoma Diagnosis and Treatment (1 paper), Protein Degradation and Inhibitors (1 paper), Ion channel regulation and function (1 paper), Chronic Myeloid Leukemia Treatments (1 paper), Blood disorders and treatments (1 paper) and Liver Diseases and Immunity (1 paper). The work is most often cited by research in Hematology (182 citations), Genetics (87 citations), Cancer Research (34 citations), Molecular Biology (132 citations) and Oncology (39 citations). Samuel Quentin has collaborated with scholars based in France, Australia and Netherlands. Frequent co-authors include Pierre Fenaux, Hervé Dombret, Nicolas Boissel, Jean Soulier, Anna Raimbault, Raphaël Itzykson, Marie Passet, Lionel Adès, Nadia Vasquez and Régis Peffault de Latour. Their work appears in journals such as Blood, Leukemia, Leukemia Research, European Journal of Human Genetics and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.