Samuel E. Marsh
Impact in
- Neurology top 0.2%
- Neuroinflammation and Neurodegeneration Mechanisms
- Neurological Disease Mechanisms and Treatments
- Biological Psychiatry top 1%
- Tryptophan and brain disorders
Papers in
-
- Neuroinflammation and Neurodegeneration Mechanisms 8
- Parkinson's Disease Mechanisms and Treatments 2
-
- Alzheimer's disease research and treatments 7
- Co-authors
- Beth Stevens (7 shared papers)Mathew Blurton‐Jones (8 shared papers)Timothy R. Hammond (2 shared papers)Adam M. H. Young (1 shared paper)Jinmiao Chen (1 shared paper)Lasse Dissing‐Olesen (1 shared paper)Alec Wysoker (1 shared paper)Robin J.M. Franklin (1 shared paper)
- Journals
- Immunity (2 papers)Trends in Immunology (1 paper)Neuron (1 paper)Cell Reports (1 paper)Alzheimer s & Dementia (1 paper)
- Partner nations
- United StatesUnited KingdomFinland
In The Last Decade
Samuel E. Marsh
13 papers receiving 2.7k citations
Samuel E. Marsh's Hit Papers
Peers
Comparison fields: 5 of 94
- Neurology 1.7k
- Biological Psychiatry 324
- Developmental Neuroscience 334
- Immunology 862
- Physiology 599
Countries citing papers authored by Samuel E. Marsh
This map shows the geographic impact of Samuel E. Marsh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel E. Marsh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel E. Marsh more than expected).
Fields of papers citing papers by Samuel E. Marsh
This network shows the impact of papers produced by Samuel E. Marsh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel E. Marsh. The network helps show where Samuel E. Marsh may publish in the future.
Co-authors
The 25 scholars most cited alongside Samuel E. Marsh, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes Hit paper breakdown → | 2018 | 1453 |
| 2 | 2016 | 318 | |
| 3 | 2019 | 250 | |
| 4 | 2020 | 174 | |
| 5 | 2017 | 144 | |
| 6 | 2017 | 133 | |
| 7 | 2012 | 104 | |
| 8 | 2017 | 49 | |
| 9 | 2018 | 39 | |
| 10 | 2022 | 19 | |
| 11 | 2017 | 13 | |
| 12 | Front and Back Flipping for Neurobiology! Developing a Hybrid Upper-Division Lab Course. | 2017 | 1 |
| 13 | 2015 | 1 | |
| 14 | 2025 | 0 | |
| 15 | 2025 | 0 | |
| 16 | 2022 | 0 |
About Samuel E. Marsh
Samuel E. Marsh is a scholar working on Neurology, Physiology, Developmental Neuroscience, Immunology and Molecular Biology, having authored 16 papers that have together received 2.7k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (8 papers), Alzheimer's disease research and treatments (7 papers), Neurogenesis and neuroplasticity mechanisms (4 papers), Tryptophan and brain disorders (3 papers), Immune Response and Inflammation (3 papers), Immune cells in cancer (3 papers), Parkinson's Disease Mechanisms and Treatments (2 papers) and Circadian rhythm and melatonin (1 paper). The work is most often cited by research in Neurology (1.7k citations), Biological Psychiatry (324 citations), Developmental Neuroscience (334 citations), Immunology (862 citations) and Physiology (599 citations). Samuel E. Marsh has collaborated with scholars based in United States, United Kingdom and Finland. Frequent co-authors include Beth Stevens, Mathew Blurton‐Jones, Timothy R. Hammond, Adam M. H. Young, Jinmiao Chen, Lasse Dissing‐Olesen, Alec Wysoker, Robin J.M. Franklin, Evan Z. Macosko and Steven A. McCarroll. Their work appears in journals such as Immunity, Trends in Immunology, Neuron, Cell Reports and Alzheimer s & Dementia.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.