Samuel Benchimol

10.7k citations
94 papers · 8.8k · 4 hit papers · h-index 46

Impact in

  • Aging top 0.5%
  • Oncology top 0.5%
    • Cancer-related Molecular Pathways

Papers in

    • Cancer-related Molecular Pathways 63
    • RNA modifications and cancer 16
    • Ubiquitin and proteasome pathways 10
    • Cell death mechanisms and regulation 9
    • DNA Repair Mechanisms 8
    • CRISPR and Genetic Engineering 7

Samuel Benchimol

94 papers receiving 8.6k citations

Samuel Benchimol's Hit Papers

Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation 2003 · 584 citations
5840+12+24Years since publication250500750

Peers

Samuel Benchimol
Comparison fields: 5 of 128
  • Aging 304
  • Oncology 4.6k
  • Biotechnology 1.2k
  • Cancer Research 1.4k
  • Molecular Biology 5.7k
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Citations per field
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Citations per year

Countries citing papers authored by Samuel Benchimol

Since Specialization
Citations

This map shows the geographic impact of Samuel Benchimol's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel Benchimol with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel Benchimol more than expected).

Fields of papers citing papers by Samuel Benchimol

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Samuel Benchimol. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel Benchimol. The network helps show where Samuel Benchimol may publish in the future.

Co-authors

The 25 scholars most cited alongside Samuel Benchimol, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Samuel Benchimol Line = papers co-authored together Samuel Benchimol links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 94 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span
Hit paper breakdown →
1998822
2
Regulation of PTEN Transcription by p53
Hit paper breakdown →
2001753
3
p53: oncogene or anti-oncogene?
Hit paper breakdown →
1990705
4
Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation
Hit paper breakdown →
2003584
5 1997313
6 1985276
7 2000274
8 1989254
9 2005248
10 1984225
11 2003166
12 2001162
13 1996161
14 1996160
15 2011158
16 1982148
17
Immortalization of rat embryo fibroblasts by the cellular p53 oncogene.
1988133
18 1993127
19 1996124
20 1991111

About Samuel Benchimol

Samuel Benchimol is a scholar working on Oncology, Molecular Biology, Genetics, Biotechnology and Cancer Research, having authored 94 papers that have together received 8.8k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (63 papers), Virus-based gene therapy research (19 papers), RNA modifications and cancer (16 papers), Cancer Research and Treatments (15 papers), Ubiquitin and proteasome pathways (10 papers), Cell death mechanisms and regulation (9 papers), DNA Repair Mechanisms (8 papers) and CRISPR and Genetic Engineering (7 papers). The work is most often cited by research in Aging (304 citations), Oncology (4.6k citations), Biotechnology (1.2k citations), Cancer Research (1.4k citations) and Molecular Biology (5.7k citations). Samuel Benchimol has collaborated with scholars based in Canada, United States and United Kingdom. Frequent co-authors include Homayoun Vaziri, David P. Lane, Yunping Lin, Weili Ma, Stephen W. Chung, Michael Mowat, Tak W. Mak, Loning Fu, Benjamin Rovinski and Rićhard P. Hill. Their work appears in journals such as Molecular and Cellular Biology, Blood, The EMBO Journal, Oncogene and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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