S Serve
Impact in
- Hematology top 2%
- Acute Myeloid Leukemia Research
- Chronic Myeloid Leukemia Treatments
- Genetics top 5%
- Chronic Lymphocytic Leukemia Research
- Myeloproliferative Neoplasms: Diagnosis and Treatment
Papers in
- Oncology 4
- Cytokine Signaling Pathways and Interactions 4
-
- Acute Myeloid Leukemia Research 2
- Chronic Myeloid Leukemia Treatments 2
- Co-authors
- Hubert Serve (5 shared papers)Hartmut Halfter (3 shared papers)Wolfgang E. Berdel (3 shared papers)R. Fenski (3 shared papers)Itaru Matsumura (2 shared papers)Masao Mizuki (2 shared papers)Karsten Kratz‐Albers (2 shared papers)Carsten Müller‐Tidow (2 shared papers)
- Journals
- Blood (2 papers)Journal of Neurochemistry (1 paper)International Journal of Oncology (1 paper)PubMed (2 papers)
- Partner nations
- GermanyUnited StatesJapan
In The Last Decade
S Serve
6 papers receiving 636 citations
Peers
Comparison fields: 5 of 46
- Hematology 383
- Genetics 167
- Oncology 148
- Molecular Biology 297
- Immunology 75
Countries citing papers authored by S Serve
This map shows the geographic impact of S Serve's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S Serve with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S Serve more than expected).
Fields of papers citing papers by S Serve
This network shows the impact of papers produced by S Serve. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S Serve. The network helps show where S Serve may publish in the future.
Co-authors
The 25 scholars most cited alongside S Serve, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2000 | 486 | |
| 2 | A truncated cyclin D1 gene encodes a stable mRNA in a human breast cancer cell line. | 1994 | 75 |
| 3 | Inhibition of proliferation and clonal growth of human breast cancer cells by interleukin 13. | 1996 | 44 |
| 4 | 2000 | 23 | |
| 5 | 2000 | 13 | |
| 6 | 1999 | 11 |
About S Serve
S Serve is a scholar working on Oncology, Hematology, Immunology, Pathology and Forensic Medicine and Pulmonary and Respiratory Medicine, having authored 6 papers that have together received 652 indexed citations. Recurring topics across this work include Cytokine Signaling Pathways and Interactions (4 papers), Acute Myeloid Leukemia Research (2 papers), Chronic Myeloid Leukemia Treatments (2 papers), Cancer Mechanisms and Therapy (1 paper), Lung Cancer Treatments and Mutations (1 paper), Transgenic Plants and Applications (1 paper), Toxin Mechanisms and Immunotoxins (1 paper) and Immune Cell Function and Interaction (1 paper). The work is most often cited by research in Hematology (383 citations), Genetics (167 citations), Oncology (148 citations), Molecular Biology (297 citations) and Immunology (75 citations). S Serve has collaborated with scholars based in Germany, United States and Japan. Frequent co-authors include Hubert Serve, Hartmut Halfter, Wolfgang E. Berdel, R. Fenski, Itaru Matsumura, Masao Mizuki, Karsten Kratz‐Albers, Carsten Müller‐Tidow, Rainer Schmidt and Joachim Kienast. Their work appears in journals such as Blood, Journal of Neurochemistry, International Journal of Oncology and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.