Robert Mandić

1.8k citations
70 papers · 1.3k · h-index 21

Impact in

Papers in

    • RNA Interference and Gene Delivery 7
    • Peptidase Inhibition and Analysis 7
    • Cancer Cells and Metastasis 4
    • HER2/EGFR in Cancer Research 4

Robert Mandić

68 papers receiving 1.3k citations

Peers

Robert Mandić
Comparison fields: 5 of 110
  • Virology 113
  • Otorhinolaryngology 97
  • Oncology 406
  • Cancer Research 200
  • Pathology and Forensic Medicine 124
Replace Carl J. DeSelm with:
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Robert Mandić relative to Carl J. DeSelm United States Carl J. DeSelm's profile →
Citations per field
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Citations per year

Countries citing papers authored by Robert Mandić

Since Specialization
Citations

This map shows the geographic impact of Robert Mandić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Mandić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Mandić more than expected).

Fields of papers citing papers by Robert Mandić

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Mandić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Mandić. The network helps show where Robert Mandić may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert Mandić, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert Mandić Line = papers co-authored together Robert Mandić links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 70 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2008108
2 200293
3 200284
4 200268
5 202264
6 200956
7 200947
8 200745
9 200543
10 200542
11 200837
12 201635
13 200135
14 200935
15 200634
16
Lymphogenic metastatic spread of auricular VX2 carcinoma in New Zealand white rabbits.
200331
17 201430
18 202029
19 201724
20 201421

About Robert Mandić

Robert Mandić is a scholar working on Molecular Biology, Oncology, Surgery, Pulmonary and Respiratory Medicine and Cancer Research, having authored 70 papers that have together received 1.3k indexed citations. Recurring topics across this work include Vascular Malformations and Hemangiomas (8 papers), RNA Interference and Gene Delivery (7 papers), Peptidase Inhibition and Analysis (7 papers), Protease and Inhibitor Mechanisms (5 papers), Cancer Cells and Metastasis (4 papers), HER2/EGFR in Cancer Research (4 papers), Salivary Gland Tumors Diagnosis and Treatment (4 papers) and Photodynamic Therapy Research Studies (4 papers). The work is most often cited by research in Virology (113 citations), Otorhinolaryngology (97 citations), Oncology (406 citations), Cancer Research (200 citations) and Pathology and Forensic Medicine (124 citations). Robert Mandić has collaborated with scholars based in Germany, United States and Finland. Frequent co-authors include Jochen A. Werner, B. Matija Peterlin, Susanne Wiegand, Thomas Linnemann, Yong‐Hui Zheng, Michael Bette, Peter Barth, Behfar Eivazi, Matthias Geyer and Afshin Teymoortash. Their work appears in journals such as Acta Oto-Laryngologica, Oral Oncology, Pharmaceutics, Clinical Cancer Research and Cancers.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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