Robert Karisch

927 citations
11 papers · 779 · h-index 9

Impact in

    • Cancer Cells and Metastasis
    • Protein Tyrosine Phosphatases
    • Redox biology and oxidative stress
    • Mitochondrial Function and Pathology
    • Epigenetics and DNA Methylation

Papers in

    • Galectins and Cancer Biology 2
    • Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2
    • Endoplasmic Reticulum Stress and Disease 2

Robert Karisch

11 papers receiving 765 citations

Peers

Robert Karisch
Comparison fields: 5 of 70
  • Oncology 181
  • Molecular Biology 472
  • Cancer Research 92
  • Cell Biology 103
  • Immunology 113
Replace Chris J. Novotny with:
Chris J. Novotny United States
Phillippa Dudley United Kingdom
Karine Malagu United Kingdom
Naniye Mallı Cetinbas United States
Claire L. McWhirter United Kingdom
Junichi Matsuzaki Japan
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Citations per field
00.5×1.7×
Chris J. Novotny · 1×
Citations per year

Countries citing papers authored by Robert Karisch

Since Specialization
Citations

This map shows the geographic impact of Robert Karisch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Karisch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Karisch more than expected).

Fields of papers citing papers by Robert Karisch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Karisch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Karisch. The network helps show where Robert Karisch may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert Karisch, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert Karisch Line = papers co-authored together Robert Karisch links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 2010195
2 2011153
3 2013105
4 200795
5 201193
6 201149
7 201238
8 200823
9 201719
10 20218
11 20131

About Robert Karisch

Robert Karisch is a scholar working on Immunology, Cell Biology, Molecular Biology, Oncology and Biotechnology, having authored 11 papers that have together received 779 indexed citations. Recurring topics across this work include Protein Tyrosine Phosphatases (5 papers), Glycosylation and Glycoproteins Research (2 papers), RNA modifications and cancer (2 papers), RNA regulation and disease (2 papers), Redox biology and oxidative stress (2 papers), Galectins and Cancer Biology (2 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). The work is most often cited by research in Oncology (181 citations), Molecular Biology (472 citations), Cancer Research (92 citations), Cell Biology (103 citations) and Immunology (113 citations). Robert Karisch has collaborated with scholars based in Canada, United States and United Kingdom. Frequent co-authors include Benjamin G. Neel, Mark Lautens, Yuan‐Qing Fang, Minerva M. Fernandez, Paul P. Taylor, Jonathan St. Germain, Lily L. Jin, Michael F. Moran, Isaac S. Harris and Arne Östman. Their work appears in journals such as Journal of Cell Science, Cell, Molecular and Cellular Biology, PROTEOMICS and FEBS Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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