Robert Gauss
Impact in
- Cell Biology top 2%
- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
- Aging top 10%
Papers in
-
- Glycosylation and Glycoproteins Research 3
- Fungal and yeast genetics research 3
- Heat shock proteins research 2
-
- Endoplasmic Reticulum Stress and Disease 7
- Cellular transport and secretion 5
- Co-authors
- Thomas Sommer (6 shared papers)Christian Hirsch (3 shared papers)Ernst Jarosch (3 shared papers)Markus Aebi (7 shared papers)Anne Spang (2 shared papers)Mark Trautwein (2 shared papers)Davis Ng (1 shared paper)Kazue Kanehara (1 shared paper)
- Journals
- Molecular Biology of the Cell (2 papers)The EMBO Journal (2 papers)Nature Cell Biology (1 paper)Nature (1 paper)Trends in Cell Biology (1 paper)
- Partner nations
- GermanySwitzerlandHungary
In The Last Decade
Robert Gauss
13 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 75
- Cell Biology 681
- Aging 25
- Molecular Biology 811
- Epidemiology 295
- Biotechnology 79
Countries citing papers authored by Robert Gauss
This map shows the geographic impact of Robert Gauss's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Gauss with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Gauss more than expected).
Fields of papers citing papers by Robert Gauss
This network shows the impact of papers produced by Robert Gauss. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Gauss. The network helps show where Robert Gauss may publish in the future.
Co-authors
The 25 scholars most cited alongside Robert Gauss, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 306 | |
| 2 | 2006 | 194 | |
| 3 | 2006 | 125 | |
| 4 | 2011 | 101 | |
| 5 | 2004 | 99 | |
| 6 | 2006 | 81 | |
| 7 | 2014 | 71 | |
| 8 | 2015 | 60 | |
| 9 | 2015 | 43 | |
| 10 | 2014 | 30 | |
| 11 | 2017 | 30 | |
| 12 | 2006 | 29 | |
| 13 | 2017 | 8 |
About Robert Gauss
Robert Gauss is a scholar working on Molecular Biology, Cell Biology, Epidemiology, Biotechnology and Immunology, having authored 13 papers that have together received 1.2k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (7 papers), Cellular transport and secretion (5 papers), Glycosylation and Glycoproteins Research (3 papers), Fungal and yeast genetics research (3 papers), Autophagy in Disease and Therapy (3 papers), Heat shock proteins research (2 papers), Galectins and Cancer Biology (2 papers) and Transgenic Plants and Applications (2 papers). The work is most often cited by research in Cell Biology (681 citations), Aging (25 citations), Molecular Biology (811 citations), Epidemiology (295 citations) and Biotechnology (79 citations). Robert Gauss has collaborated with scholars based in Germany, Switzerland and Hungary. Frequent co-authors include Thomas Sommer, Christian Hirsch, Ernst Jarosch, Markus Aebi, Anne Spang, Mark Trautwein, Davis Ng, Kazue Kanehara, Pedro Carvalho and Jörn Dengjel. Their work appears in journals such as Molecular Biology of the Cell, The EMBO Journal, Nature Cell Biology, Nature and Trends in Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.