Ratika Kunder
Impact in
-
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
-
- Cancer Mechanisms and Therapy
Papers in
-
- Circular RNAs in diseases 3
- Cancer therapeutics and mechanisms 1
- Mechanisms of cancer metastasis 1
- Extracellular vesicles in disease 1
-
- MicroRNA in disease regulation 3
- Cancer-related molecular mechanisms research 2
- Co-authors
- Neelam Shirsat (3 shared papers)Atul Goel (3 shared papers)Aliasgar Moiyadi (3 shared papers)Sadhana Kannan (2 shared papers)Rajiv Sarin (1 shared paper)Asha Shenoy (1 shared paper)Santosh Noronha (1 shared paper)Dai Horiuchi (2 shared papers)
- Journals
- Acta Neuropathologica Communications (1 paper)Nature Medicine (1 paper)Neuro-Oncology (1 paper)Cell chemical biology (1 paper)Journal of Cancer Research and Therapeutics (1 paper)
- Partner nations
- IndiaUnited StatesCanada
In The Last Decade
Ratika Kunder
5 papers receiving 329 citations
Peers
Comparison fields: 5 of 37
- Cancer Research 145
- Pathology and Forensic Medicine 83
- Genetics 49
- Molecular Biology 256
- Oncology 73
Countries citing papers authored by Ratika Kunder
This map shows the geographic impact of Ratika Kunder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ratika Kunder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ratika Kunder more than expected).
Fields of papers citing papers by Ratika Kunder
This network shows the impact of papers produced by Ratika Kunder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ratika Kunder. The network helps show where Ratika Kunder may publish in the future.
Co-authors
The 25 scholars most cited alongside Ratika Kunder, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 122 | |
| 2 | 2010 | 104 | |
| 3 | 2013 | 69 | |
| 4 | 2019 | 21 | |
| 5 | 2021 | 14 |
About Ratika Kunder
Ratika Kunder is a scholar working on Molecular Biology, Cancer Research, Pathology and Forensic Medicine, Oncology and Infectious Diseases, having authored 5 papers that have together received 330 indexed citations. Recurring topics across this work include MicroRNA in disease regulation (3 papers), Circular RNAs in diseases (3 papers), Cancer-related molecular mechanisms research (2 papers), Cancer Mechanisms and Therapy (2 papers), Peptidase Inhibition and Analysis (2 papers), Cancer therapeutics and mechanisms (1 paper), Mechanisms of cancer metastasis (1 paper) and Extracellular vesicles in disease (1 paper). The work is most often cited by research in Cancer Research (145 citations), Pathology and Forensic Medicine (83 citations), Genetics (49 citations), Molecular Biology (256 citations) and Oncology (73 citations). Ratika Kunder has collaborated with scholars based in India, United States and Canada. Frequent co-authors include Neelam Shirsat, Atul Goel, Aliasgar Moiyadi, Sadhana Kannan, Rajiv Sarin, Asha Shenoy, Santosh Noronha, Dai Horiuchi, Devon A. Lawson and Olga Momčilović. Their work appears in journals such as Acta Neuropathologica Communications, Nature Medicine, Neuro-Oncology, Cell chemical biology and Journal of Cancer Research and Therapeutics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.