Rainer Schäfer

19 papers receiving 538 citations

Peers

Rainer Schäfer
Comparison fields: 5 of 70
  • Physiology 294
  • Endocrine and Autonomic Systems 49
  • Cellular and Molecular Neuroscience 112
  • Molecular Biology 302
  • Sensory Systems 17
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Citations per year

Countries citing papers authored by Rainer Schäfer

Since Specialization
Citations

This map shows the geographic impact of Rainer Schäfer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rainer Schäfer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rainer Schäfer more than expected).

Fields of papers citing papers by Rainer Schäfer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rainer Schäfer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rainer Schäfer. The network helps show where Rainer Schäfer may publish in the future.

Co-authors

The 25 scholars most cited alongside Rainer Schäfer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Rainer Schäfer Line = papers co-authored together Rainer Schäfer links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 22 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2003112
2 200786
3 199043
4 200039
5 200533
6 199533
7 197628
8 198823
9 199521
10 199120
11 199720
12
Establishment of a cellular assay system for G protein-linked receptors: coupling of human NK2 and 5-HT2 receptors to phospholipase C activates a luciferase reporter gene.
199320
13 199019
14 197617
15 200612
16 200111
17 19999
18 20069
19
Johann Gottlieb Fichtes 'Grundlage der gesamten Wissenschaftslehre von 1794'
20061
20 20111

About Rainer Schäfer

Rainer Schäfer is a scholar working on Molecular Biology, Physiology, Cellular and Molecular Neuroscience, Organic Chemistry and Physiology, having authored 22 papers that have together received 557 indexed citations. Recurring topics across this work include Adenosine and Purinergic Signaling (9 papers), Receptor Mechanisms and Signaling (7 papers), Protein Kinase Regulation and GTPase Signaling (3 papers), Neuroscience and Neuropharmacology Research (2 papers), Synthesis and Properties of Aromatic Compounds (2 papers), Electrolyte and hormonal disorders (2 papers), Kantian Philosophy and Modern Interpretations (2 papers) and Neuropeptides and Animal Physiology (2 papers). The work is most often cited by research in Physiology (294 citations), Endocrine and Autonomic Systems (49 citations), Cellular and Molecular Neuroscience (112 citations), Molecular Biology (302 citations) and Sensory Systems (17 citations). Rainer Schäfer has collaborated with scholars based in Germany, United States and Austria. Frequent co-authors include Georg Reiser, Fariba Sedehizade, Tobias Welte, Theodor Hanck, Mohan E. Tulapurkar, Christian Vöhringer, Matthias U. Kassack, Rolf Stricker, Shigeo Tanimoto and Irene Schulz. Their work appears in journals such as Biochemical Journal, Biochemical Pharmacology, Biochemical and Biophysical Research Communications, British Journal of Pharmacology and American Journal of Physiology-Lung Cellular and Molecular Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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