Quentin Nevers
Impact in
- Virology top 10%
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- SARS-CoV-2 and COVID-19 Research
- Viral Infections and Vectors
Papers in
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- Signaling Pathways in Disease 2
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- Influenza Virus Research Studies 3
- Hepatitis B Virus Studies 2
- Co-authors
- Yves Gaudin (3 shared papers)Cécile Lagaudrière‐Gesbert (3 shared papers)Aurélie Albertini (2 shared papers)Abdelhakim Ahmed‐Belkacem (5 shared papers)Rozenn Brillet (5 shared papers)Jean–Michel Pawlotsky (5 shared papers)Nazim Ahnou (4 shared papers)Olivier Schwartz (2 shared papers)
- Journals
- Antimicrobial Agents and Chemotherapy (3 papers)PLoS Pathogens (2 papers)Journal of Virology (1 paper)Cell Reports (1 paper)Veterinary Research (1 paper)
- Partner nations
- FranceUnited States
In The Last Decade
Quentin Nevers
12 papers receiving 405 citations
Peers
Comparison fields: 5 of 67
- Virology 48
- Infectious Diseases 102
- Obstetrics and Gynecology 37
- Immunology 104
- Molecular Biology 195
Countries citing papers authored by Quentin Nevers
This map shows the geographic impact of Quentin Nevers's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Quentin Nevers with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Quentin Nevers more than expected).
Fields of papers citing papers by Quentin Nevers
This network shows the impact of papers produced by Quentin Nevers. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Quentin Nevers. The network helps show where Quentin Nevers may publish in the future.
Co-authors
The 25 scholars most cited alongside Quentin Nevers, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 114 | |
| 2 | 2016 | 68 | |
| 3 | 2020 | 65 | |
| 4 | 2020 | 53 | |
| 5 | 2019 | 39 | |
| 6 | 2022 | 20 | |
| 7 | 2023 | 17 | |
| 8 | 2018 | 11 | |
| 9 | 2022 | 10 | |
| 10 | 2020 | 7 | |
| 11 | 2024 | 3 | |
| 12 | 2024 | 2 | |
| 13 | 2025 | 0 |
About Quentin Nevers
Quentin Nevers is a scholar working on Molecular Biology, Epidemiology, Immunology, Infectious Diseases and Virology, having authored 13 papers that have together received 409 indexed citations. Recurring topics across this work include Rabies epidemiology and control (3 papers), interferon and immune responses (3 papers), Influenza Virus Research Studies (3 papers), Toxin Mechanisms and Immunotoxins (2 papers), Hepatitis B Virus Studies (2 papers), Signaling Pathways in Disease (2 papers), Viral Infections and Immunology Research (2 papers) and Hepatitis C virus research (2 papers). The work is most often cited by research in Virology (48 citations), Infectious Diseases (102 citations), Obstetrics and Gynecology (37 citations), Immunology (104 citations) and Molecular Biology (195 citations). Quentin Nevers has collaborated with scholars based in France and United States. Frequent co-authors include Yves Gaudin, Cécile Lagaudrière‐Gesbert, Aurélie Albertini, Abdelhakim Ahmed‐Belkacem, Rozenn Brillet, Jean–Michel Pawlotsky, Nazim Ahnou, Olivier Schwartz, Françoise Porrot and Julian Buchrieser. Their work appears in journals such as Antimicrobial Agents and Chemotherapy, PLoS Pathogens, Journal of Virology, Cell Reports and Veterinary Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.