Peter J. Lyons

733 citations
20 papers · 522 · h-index 14

Impact in

    • Microtubule and mitosis dynamics
    • Ubiquitin and proteasome pathways
    • Signaling Pathways in Disease
    • Peroxisome Proliferator-Activated Receptors

Papers in

    • Peroxisome Proliferator-Activated Receptors 4
    • Signaling Pathways in Disease 3
    • RNA Research and Splicing 2
    • Receptor Mechanisms and Signaling 2
    • Peptidase Inhibition and Analysis 7

Peter J. Lyons

19 papers receiving 515 citations

Peers

Peter J. Lyons
Comparison fields: 5 of 82
  • Cell Biology 98
  • Molecular Biology 336
  • Cancer Research 69
  • Oncology 100
  • Genetics 95
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Chadi Zakaria Canada
Xiaoduo Xie China
David Wan‐Cheng Li China
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Citations per field
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Citations per year

Countries citing papers authored by Peter J. Lyons

Since Specialization
Citations

This map shows the geographic impact of Peter J. Lyons's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter J. Lyons with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter J. Lyons more than expected).

Fields of papers citing papers by Peter J. Lyons

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter J. Lyons. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter J. Lyons. The network helps show where Peter J. Lyons may publish in the future.

Co-authors

The 25 scholars most cited alongside Peter J. Lyons, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Peter J. Lyons Line = papers co-authored together Peter J. Lyons links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 2001113
2 201173
3 200846
4 200737
5 201334
6 201133
7 201031
8 201127
9 201323
10 201019
11 201217
12 201816
13 200613
14 200413
15 201112
16 20236
17 20204
18 20184
19 20221
20 20240

About Peter J. Lyons

Peter J. Lyons is a scholar working on Molecular Biology, Oncology, Cell Biology, Cellular and Molecular Neuroscience and Genetics, having authored 20 papers that have together received 522 indexed citations. Recurring topics across this work include Peptidase Inhibition and Analysis (7 papers), Peroxisome Proliferator-Activated Receptors (4 papers), Signaling Pathways in Disease (3 papers), Neuropeptides and Animal Physiology (3 papers), RNA Research and Splicing (2 papers), Receptor Mechanisms and Signaling (2 papers), Microtubule and mitosis dynamics (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). The work is most often cited by research in Cell Biology (98 citations), Molecular Biology (336 citations), Cancer Research (69 citations), Oncology (100 citations) and Genetics (95 citations). Peter J. Lyons has collaborated with scholars based in United States, Canada and Japan. Frequent co-authors include Lloyd D. Fricker, Jungsil Ro, Aleixo M. Muise, Sung Woo Kim, Myrasol Callaway, Matthew R. Sapio, Juan Sironi, Koji Ikegami, Iryna Berezniuk and Hui Xiao. Their work appears in journals such as Journal of Biological Chemistry, PLoS ONE, Human Mutation, Scientific Reports and Frontiers in Molecular Biosciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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