Nil Ege
Impact in
- Cell Biology top 2%
- Hippo pathway signaling and YAP/TAZ
- Cellular Mechanics and Interactions
- Microtubule and mitosis dynamics
- Immunology and Allergy top 5%
- Cell Adhesion Molecules Research
Papers in
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- Protein Degradation and Inhibitors 1
- Wnt/β-catenin signaling in development and cancer 1
- Ubiquitin and proteasome pathways 1
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- Hippo pathway signaling and YAP/TAZ 2
- Cellular Mechanics and Interactions 2
- Co-authors
- Steven Hooper (2 shared papers)Robert P. Jenkins (2 shared papers)Erik Sahai (2 shared papers)Fernando Calvo (1 shared paper)A. Grande-García (1 shared paper)Kevin J. Harrington (1 shared paper)Peter Williamson (1 shared paper)Guillaume Charras (1 shared paper)
- Journals
- Nature Cell Biology (1 paper)Cell Systems (1 paper)The Journal of Immunology (1 paper)Cell chemical biology (1 paper)
- Partner nations
- United KingdomIsraelFrance
In The Last Decade
Nil Ege
4 papers receiving 1.2k citations
Nil Ege's Hit Papers
Peers
Comparison fields: 5 of 78
- Cell Biology 724
- Immunology and Allergy 104
- Oncology 441
- Cancer Research 168
- Molecular Biology 593
Countries citing papers authored by Nil Ege
This map shows the geographic impact of Nil Ege's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nil Ege with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nil Ege more than expected).
Fields of papers citing papers by Nil Ege
This network shows the impact of papers produced by Nil Ege. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nil Ege. The network helps show where Nil Ege may publish in the future.
Co-authors
The 20 scholars most cited alongside Nil Ege, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Mechanotransduction and YAP-dependent matrix remodelling is required for the generation and maintenance of cancer-associated fibroblasts Hit paper breakdown → | 2013 | 1076 |
| 2 | 2018 | 100 | |
| 3 | 2011 | 59 | |
| 4 | 2021 | 19 |
About Nil Ege
Nil Ege is a scholar working on Molecular Biology, Cell Biology, Hematology, Immunology and Plant Science, having authored 4 papers that have together received 1.3k indexed citations. Recurring topics across this work include Hippo pathway signaling and YAP/TAZ (2 papers), Cellular Mechanics and Interactions (2 papers), Protein Degradation and Inhibitors (1 paper), Plant Surface Properties and Treatments (1 paper), T-cell and B-cell Immunology (1 paper), Multiple Myeloma Research and Treatments (1 paper), Wnt/β-catenin signaling in development and cancer (1 paper) and Ubiquitin and proteasome pathways (1 paper). The work is most often cited by research in Cell Biology (724 citations), Immunology and Allergy (104 citations), Oncology (441 citations), Cancer Research (168 citations) and Molecular Biology (593 citations). Nil Ege has collaborated with scholars based in United Kingdom, Israel and France. Frequent co-authors include Steven Hooper, Robert P. Jenkins, Erik Sahai, Fernando Calvo, A. Grande-García, Kevin J. Harrington, Peter Williamson, Guillaume Charras, Emad Moeendarbary and C. T. Foster. Their work appears in journals such as Nature Cell Biology, Cell Systems, The Journal of Immunology and Cell chemical biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.