Nathan Snyder

458 citations
11 papers · 289 · 1 hit paper · h-index 6

Impact in

    • Ubiquitin and proteasome pathways
    • Histone Deacetylase Inhibitors Research
    • Protein Degradation and Inhibitors
    • RNA modifications and cancer
    • Endoplasmic Reticulum Stress and Disease

Papers in

    • Ubiquitin and proteasome pathways 4
    • Ion channel regulation and function 1
    • Endoplasmic Reticulum Stress and Disease 5
    • Cellular transport and secretion 2

Nathan Snyder

8 papers receiving 288 citations

Nathan Snyder's Hit Papers

Deubiquitinating enzymes (DUBs): Regulation, homeostasis, and oxidative stress response 2021 · 186 citations
1860+1+3Years since publication50100150

Peers

Nathan Snyder
Comparison fields: 5 of 63
  • Molecular Biology 226
  • Cell Biology 48
  • Oncology 56
  • Aging 3
  • Immunology 28
Replace Pablo Gallego with:
Pablo Gallego Spain
J.L. Schipper United States
Xingrun Zhang China
Yogesh Ostwal Germany
Jonah Beenstock Israel
Xiomaris M. Cotto-Rios United States
Tatsuya Yano Japan
Shahlo Тurdikulova Uzbekistan
Helene Klug Germany
Christopher R. Horne Australia
Nathan Snyder relative to Pablo Gallego Spain Pablo Gallego's profile →
Citations per field
00.5×1.5×2.3×
Pablo Gallego · 1×
Citations per year

Countries citing papers authored by Nathan Snyder

Since Specialization
Citations

This map shows the geographic impact of Nathan Snyder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan Snyder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan Snyder more than expected).

Fields of papers citing papers by Nathan Snyder

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan Snyder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan Snyder. The network helps show where Nathan Snyder may publish in the future.

Co-authors

The 25 scholars most cited alongside Nathan Snyder, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Nathan Snyder Line = papers co-authored together Nathan Snyder links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1
Deubiquitinating enzymes (DUBs): Regulation, homeostasis, and oxidative stress response
Hit paper breakdown →
2021186
2 202223
3 201823
4 201720
5 201917
6 201916
7 20243
8 20241
9 20240
10 20240
11 20250

About Nathan Snyder

Nathan Snyder is a scholar working on Molecular Biology, Cell Biology, Pathology and Forensic Medicine, Genetics and Epidemiology, having authored 11 papers that have together received 289 indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (5 papers), Ubiquitin and proteasome pathways (4 papers), Genetic factors in colorectal cancer (3 papers), Cellular transport and secretion (2 papers), Pancreatic function and diabetes (1 paper), Ion channel regulation and function (1 paper), Genetic Associations and Epidemiology (1 paper) and Cancer Genomics and Diagnostics (1 paper). The work is most often cited by research in Molecular Biology (226 citations), Cell Biology (48 citations), Oncology (56 citations), Aging (3 citations) and Immunology (28 citations). Nathan Snyder has collaborated with scholars based in United States, Switzerland and China. Frequent co-authors include Gustavo M. Silva, Kyle W. Cunningham, Linghuo Jiang, Adam Kim, Junjun Wang, Timothy A. Reinhardt, Mitchell V. Palmer, Carlos Evangelista, Christopher P. Stefan and Alberto Bartesaghi. Their work appears in journals such as Journal of Biological Chemistry, Journal of Clinical Oncology, G3 Genes Genomes Genetics, JAMA Oncology and Prostate Cancer and Prostatic Diseases.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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