William B. Isaacs
Impact in
- Cancer Research top 0.05%
- Cancer, Lipids, and Metabolism
- Cancer, Hypoxia, and Metabolism
- Cancer-related molecular mechanisms research
- Pulmonary and Respiratory Medicine top 0.02%
- Prostate Cancer Treatment and Research
- Prostate Cancer Diagnosis and Treatment
Papers in
-
- Prostate Cancer Treatment and Research 153
- Prostate Cancer Diagnosis and Treatment 79
-
- Cancer-related gene regulation 41
- Epigenetics and DNA Methylation 35
- Co-authors
- Angelo M. De Marzo (81 shared papers)William G. Nelson (33 shared papers)G. Steven Bova (60 shared papers)Charles M. Ewing (44 shared papers)Jonathan I. Epstein (20 shared papers)Jianfeng Xu (69 shared papers)Jun Luo (29 shared papers)Elizabeth A. Platz (41 shared papers)
- Journals
- The Prostate (75 papers)The Journal of Urology (34 papers)Cancer Research (17 papers)Cancer Epidemiology Biomarkers & Prevention (17 papers)Clinical Cancer Research (13 papers)
- Partner nations
- United StatesSwedenUnited Kingdom
In The Last Decade
William B. Isaacs
378 papers receiving 33.2k citations
William B. Isaacs's Hit Papers
Peers
Comparison fields: 5 of 192
- Cancer Research 8.2k
- Pulmonary and Respiratory Medicine 13.4k
- Oncology 5.8k
- Molecular Biology 15.4k
- Genetics 3.7k
Countries citing papers authored by William B. Isaacs
This map shows the geographic impact of William B. Isaacs's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William B. Isaacs with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William B. Isaacs more than expected).
Fields of papers citing papers by William B. Isaacs
This network shows the impact of papers produced by William B. Isaacs. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William B. Isaacs. The network helps show where William B. Isaacs may publish in the future.
Co-authors
The 25 scholars most cited alongside William B. Isaacs, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 383 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Overexpression of hypoxia-inducible factor 1alpha in common human cancers and their metastases. Hit paper breakdown → | 1999 | 1976 |
| 2 | Inflammation in prostate carcinogenesis Hit paper breakdown → | 2007 | 1294 |
| 3 | DD3: a new prostate-specific gene, highly overexpressed in prostate cancer. Hit paper breakdown → | 1999 | 865 |
| 4 | Ligand-Independent Androgen Receptor Variants Derived from Splicing of Cryptic Exons Signify Hormone-Refractory Prostate Cancer Hit paper breakdown → | 2008 | 859 |
| 5 | Prostate Cancer Hit paper breakdown → | 2003 | 809 |
| 6 | Cytidine methylation of regulatory sequences near the pi-class glutathione S-transferase gene accompanies human prostatic carcinogenesis. Hit paper breakdown → | 1994 | 703 |
| 7 | E-cadherin expression is silenced by DNA hypermethylation in human breast and prostate carcinomas. Hit paper breakdown → | 1995 | 701 |
| 8 | DPC4 gene in various tumor types. Hit paper breakdown → | 1996 | 590 |
| 9 | Frequent inactivation of PTEN/MMAC1 in primary prostate cancer. Hit paper breakdown → | 1997 | 585 |
| 10 | Inactivation of the tumor suppressor PTEN/MMAC1 in advanced human prostate cancer through loss of expression Hit paper breakdown → | 1998 | 555 |
| 11 | Expression of the cellular adhesion molecule E-cadherin is reduced or absent in high-grade prostate cancer. Hit paper breakdown → | 1992 | 533 |
| 12 | 2010 | 469 | |
| 13 | 1993 | 468 | |
| 14 | Decreased E-cadherin expression is associated with poor prognosis in patients with prostate cancer. | 1994 | 466 |
| 15 | Distinct Transcriptional Programs Mediated by the Ligand-Dependent Full-Length Androgen Receptor and Its Splice Variants in Castration-Resistant Prostate Cancer Hit paper breakdown → | 2012 | 463 |
| 16 | 2004 | 434 | |
| 17 | 1990 | 406 | |
| 18 | Wild-type p53 suppresses growth of human prostate cancer cells containing mutant p53 alleles. | 1991 | 399 |
| 19 | Interfocal heterogeneity of PTEN/MMAC1 gene alterations in multiple metastatic prostate cancer tissues. | 1998 | 390 |
| 20 | Establishment and characterization of seven dunning rat prostatic cancer cell lines and their use in developing methods for predicting metastatic abilities of prostatic cancers Hit paper breakdown → | 1986 | 374 |
About William B. Isaacs
William B. Isaacs is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology, Cancer Research, Genetics and Oncology, having authored 383 papers that have together received 34.4k indexed citations. Recurring topics across this work include Prostate Cancer Treatment and Research (153 papers), Prostate Cancer Diagnosis and Treatment (79 papers), Cancer-related gene regulation (41 papers), Epigenetics and DNA Methylation (35 papers), Cancer Genomics and Diagnostics (27 papers), Cancer, Lipids, and Metabolism (26 papers), Genetic factors in colorectal cancer (24 papers) and Genetic Associations and Epidemiology (21 papers). The work is most often cited by research in Cancer Research (8.2k citations), Pulmonary and Respiratory Medicine (13.4k citations), Oncology (5.8k citations), Molecular Biology (15.4k citations) and Genetics (3.7k citations). William B. Isaacs has collaborated with scholars based in United States, Sweden and United Kingdom. Frequent co-authors include Angelo M. De Marzo, William G. Nelson, G. Steven Bova, Charles M. Ewing, Jonathan I. Epstein, Jianfeng Xu, Jun Luo, Elizabeth A. Platz, Jack A. Schalken and Henrik Grönberg. Their work appears in journals such as The Prostate, The Journal of Urology, Cancer Research, Cancer Epidemiology Biomarkers & Prevention and Clinical Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.