Nadja Schulz
Impact in
- Aging top 10%
- Genetics, Aging, and Longevity in Model Organisms
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- Telomeres, Telomerase, and Senescence
- Adipose Tissue and Metabolism
Papers in
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- Metabolism, Diabetes, and Cancer 3
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- Adipose Tissue and Metabolism 4
- Diet and metabolism studies 2
- Co-authors
- Annette Schürmann (7 shared papers)Olov Andersson (3 shared papers)Doreen Kuhlow (1 shared paper)K. Lenhard Rudolph (1 shared paper)Sandra Weimer (1 shared paper)Guido von Figura (1 shared paper)Simone Florian (1 shared paper)Kim Zarse (1 shared paper)
- Journals
- Molecular and Cellular Biology (1 paper)The EMBO Journal (1 paper)Endocrinology (1 paper)Aging (1 paper)Laboratory Animals (1 paper)
- Partner nations
- GermanySwedenUnited States
In The Last Decade
Nadja Schulz
12 papers receiving 516 citations
Peers
Comparison fields: 5 of 77
- Aging 27
- Physiology 168
- Endocrinology, Diabetes and Metabolism 99
- Endocrine and Autonomic Systems 30
- Genetics 114
Countries citing papers authored by Nadja Schulz
This map shows the geographic impact of Nadja Schulz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadja Schulz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadja Schulz more than expected).
Fields of papers citing papers by Nadja Schulz
This network shows the impact of papers produced by Nadja Schulz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadja Schulz. The network helps show where Nadja Schulz may publish in the future.
Co-authors
The 25 scholars most cited alongside Nadja Schulz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2010 | 103 | |
| 2 | 2014 | 86 | |
| 3 | 2016 | 70 | |
| 4 | 2009 | 61 | |
| 5 | 2016 | 43 | |
| 6 | 2011 | 34 | |
| 7 | 2016 | 28 | |
| 8 | 2021 | 26 | |
| 9 | 2013 | 22 | |
| 10 | 2007 | 20 | |
| 11 | 2015 | 15 | |
| 12 | 2013 | 14 | |
| 13 | 2018 | 0 |
About Nadja Schulz
Nadja Schulz is a scholar working on Molecular Biology, Physiology, Surgery, Endocrinology, Diabetes and Metabolism and Cell Biology, having authored 13 papers that have together received 522 indexed citations. Recurring topics across this work include Pancreatic function and diabetes (5 papers), Adipose Tissue and Metabolism (4 papers), Metabolism, Diabetes, and Cancer (3 papers), Diabetes Management and Research (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Lipid metabolism and biosynthesis (2 papers), Diet and metabolism studies (2 papers) and Diet, Metabolism, and Disease (1 paper). The work is most often cited by research in Aging (27 citations), Physiology (168 citations), Endocrinology, Diabetes and Metabolism (99 citations), Endocrine and Autonomic Systems (30 citations) and Genetics (114 citations). Nadja Schulz has collaborated with scholars based in Germany, Sweden and United States. Frequent co-authors include Annette Schürmann, Olov Andersson, Doreen Kuhlow, K. Lenhard Rudolph, Sandra Weimer, Guido von Figura, Simone Florian, Kim Zarse, Klaus J. Petzke and Andreas Pfeiffer. Their work appears in journals such as Molecular and Cellular Biology, The EMBO Journal, Endocrinology, Aging and Laboratory Animals.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.