Moritz M. Hettich
Impact in
- Aging top 5%
- Genetics, Aging, and Longevity in Model Organisms
- Physiology top 10%
- Alzheimer's disease research and treatments
- Adipose Tissue and Metabolism
Papers in
-
- Metabolism, Diabetes, and Cancer 5
- Wnt/β-catenin signaling in development and cancer 2
- Ubiquitin and proteasome pathways 1
-
- Alzheimer's disease research and treatments 5
- Co-authors
- Sybille Krauß (5 shared papers)Wilhelm Krone (5 shared papers)Michael Udelhoven (5 shared papers)Dan Ehninger (6 shared papers)Markus Schubert (4 shared papers)S Freude (4 shared papers)Jens C. Brüning (3 shared papers)Frank Matthes (4 shared papers)
In The Last Decade
Moritz M. Hettich
12 papers receiving 777 citations
Peers
Comparison fields: 5 of 71
- Aging 62
- Physiology 307
- Geriatrics and Gerontology 37
- Biological Psychiatry 26
- Developmental Neuroscience 41
Countries citing papers authored by Moritz M. Hettich
This map shows the geographic impact of Moritz M. Hettich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Moritz M. Hettich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Moritz M. Hettich more than expected).
Fields of papers citing papers by Moritz M. Hettich
This network shows the impact of papers produced by Moritz M. Hettich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Moritz M. Hettich. The network helps show where Moritz M. Hettich may publish in the future.
Co-authors
The 25 scholars most cited alongside Moritz M. Hettich, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 187 | |
| 2 | 2014 | 125 | |
| 3 | 2009 | 90 | |
| 4 | 2017 | 74 | |
| 5 | 2014 | 73 | |
| 6 | 2011 | 59 | |
| 7 | 2008 | 53 | |
| 8 | 2018 | 38 | |
| 9 | 2012 | 34 | |
| 10 | 2015 | 31 | |
| 11 | 2009 | 17 | |
| 12 | 2015 | 4 |
About Moritz M. Hettich
Moritz M. Hettich is a scholar working on Molecular Biology, Physiology, Cellular and Molecular Neuroscience, Endocrinology, Diabetes and Metabolism and Cell Biology, having authored 12 papers that have together received 785 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (5 papers), Metabolism, Diabetes, and Cancer (5 papers), Genetics and Neurodevelopmental Disorders (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Growth Hormone and Insulin-like Growth Factors (2 papers), Wnt/β-catenin signaling in development and cancer (2 papers), Neurogenesis and neuroplasticity mechanisms (1 paper) and Ubiquitin and proteasome pathways (1 paper). The work is most often cited by research in Aging (62 citations), Physiology (307 citations), Geriatrics and Gerontology (37 citations), Biological Psychiatry (26 citations) and Developmental Neuroscience (41 citations). Moritz M. Hettich has collaborated with scholars based in Germany, Japan and Austria. Frequent co-authors include Sybille Krauß, Wilhelm Krone, Michael Udelhoven, Dan Ehninger, Markus Schubert, S Freude, Jens C. Brüning, Frank Matthes, Uschi Leeser and Donato A. Di Monte. Their work appears in journals such as Neural Plasticity, Journal of Neurochemistry, The FASEB Journal, Journal of Biological Chemistry and Alzheimer s & Dementia.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.