Milesa Simic
Impact in
- Developmental Neuroscience top 2%
- Neurogenesis and neuroplasticity mechanisms
- Neurology top 10%
- Neuroinflammation and Neurodegeneration Mechanisms
Papers in
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- Neurogenesis and neuroplasticity mechanisms 2
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- Neuroinflammation and Neurodegeneration Mechanisms 1
- Vagus Nerve Stimulation Research 1
- Co-authors
- Masato Nakafuku (2 shared papers)Laura B. Chipman (1 shared paper)Thomas A. Reh (1 shared paper)Matthew S. Wilken (1 shared paper)Kristin E. Cox (1 shared paper)Nikolas L. Jorstad (1 shared paper)Yumi Ueki (1 shared paper)Mélanie Lebel (1 shared paper)
- Journals
- Nature Communications (2 papers)Cell Reports (1 paper)Neuron (1 paper)Journal of Neuroinflammation (1 paper)Proceedings of the National Academy of Sciences (1 paper)
- Partner nations
- FranceGermanyUnited States
In The Last Decade
Milesa Simic
6 papers receiving 544 citations
Peers
Comparison fields: 5 of 54
- Developmental Neuroscience 183
- Neurology 94
- Cellular and Molecular Neuroscience 130
- Molecular Biology 296
- Immunology 80
Countries citing papers authored by Milesa Simic
This map shows the geographic impact of Milesa Simic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Milesa Simic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Milesa Simic more than expected).
Fields of papers citing papers by Milesa Simic
This network shows the impact of papers produced by Milesa Simic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Milesa Simic. The network helps show where Milesa Simic may publish in the future.
Co-authors
The 25 scholars most cited alongside Milesa Simic, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 195 | |
| 2 | 2014 | 167 | |
| 3 | 2022 | 73 | |
| 4 | 2022 | 49 | |
| 5 | 2020 | 44 | |
| 6 | 2023 | 23 |
About Milesa Simic
Milesa Simic is a scholar working on Developmental Neuroscience, Neurology, Immunology, Molecular Biology and Cancer Research, having authored 6 papers that have together received 551 indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (2 papers), Axon Guidance and Neuronal Signaling (1 paper), IL-33, ST2, and ILC Pathways (1 paper), Renal and related cancers (1 paper), Zebrafish Biomedical Research Applications (1 paper), Neuroinflammation and Neurodegeneration Mechanisms (1 paper), Vagus Nerve Stimulation Research (1 paper) and Retinal Development and Disorders (1 paper). The work is most often cited by research in Developmental Neuroscience (183 citations), Neurology (94 citations), Cellular and Molecular Neuroscience (130 citations), Molecular Biology (296 citations) and Immunology (80 citations). Milesa Simic has collaborated with scholars based in France, Germany and United States. Frequent co-authors include Masato Nakafuku, Laura B. Chipman, Thomas A. Reh, Matthew S. Wilken, Kristin E. Cox, Nikolas L. Jorstad, Yumi Ueki, Mélanie Lebel, Jimena Andersen and Noelia Urbán. Their work appears in journals such as Nature Communications, Cell Reports, Neuron, Journal of Neuroinflammation and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.