Michael Woodside

1.6k citations
15 papers · 1.3k · h-index 14

Impact in

Papers in

    • Ion Transport and Channel Regulation 9
    • Ion channel regulation and function 7
    • Lipid Membrane Structure and Behavior 2
    • Receptor Mechanisms and Signaling 2
    • ATP Synthase and ATPases Research 1
    • Protein Degradation and Inhibitors 1
    • Pancreatic function and diabetes 4

Michael Woodside

15 papers receiving 1.3k citations

Peers

Michael Woodside
Comparison fields: 5 of 100
  • Cell Biology 316
  • Immunology and Allergy 87
  • Molecular Biology 924
  • Physiology 46
  • Cellular and Molecular Neuroscience 164
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Citations per field
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Citations per year

Countries citing papers authored by Michael Woodside

Since Specialization
Citations

This map shows the geographic impact of Michael Woodside's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Woodside with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Woodside more than expected).

Fields of papers citing papers by Michael Woodside

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Woodside. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Woodside. The network helps show where Michael Woodside may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael Woodside, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael Woodside Line = papers co-authored together Michael Woodside links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 1997334
2 1992196
3 1993162
4 1997128
5 1991101
6 199488
7 199986
8 201057
9 199552
10 199740
11 199932
12 199726
13 199921
14 199416
15 19976

About Michael Woodside

Michael Woodside is a scholar working on Molecular Biology, Surgery, Cell Biology, Cellular and Molecular Neuroscience and Ecology, having authored 15 papers that have together received 1.3k indexed citations. Recurring topics across this work include Ion Transport and Channel Regulation (9 papers), Ion channel regulation and function (7 papers), Pancreatic function and diabetes (4 papers), Lipid Membrane Structure and Behavior (2 papers), Receptor Mechanisms and Signaling (2 papers), ATP Synthase and ATPases Research (1 paper), Endoplasmic Reticulum Stress and Disease (1 paper) and Protein Degradation and Inhibitors (1 paper). The work is most often cited by research in Cell Biology (316 citations), Immunology and Allergy (87 citations), Molecular Biology (924 citations), Physiology (46 citations) and Cellular and Molecular Neuroscience (164 citations). Michael Woodside has collaborated with scholars based in Canada, United States and Finland. Frequent co-authors include Sergio Grinstein, Nicolas Demaurex, Jacques Pouysségur, Shoukat Dedhar, John Orlowski, René St‐Arnaud, Claude Sardet, Marc G. Coppolino, Daniela Rotin and Gregory P. Downey. Their work appears in journals such as Journal of Biological Chemistry, Biochemical Society Transactions, Nature, European Journal of Biochemistry and American Journal of Physiology-Cell Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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