Michael Kacik
Impact in
- Sensory Systems top 1%
- Ion Channels and Receptors
- Physiology top 10%
- Nitric Oxide and Endothelin Effects
- Erythrocyte Function and Pathophysiology
Papers in
-
- Ion Channels and Receptors 3
-
- Nitric Oxide and Endothelin Effects 3
- Co-authors
- Joachim Hoyer (5 shared papers)Ivica Grgic (4 shared papers)Ralf Köhler (4 shared papers)Willm‐Thomas Heyken (2 shared papers)Christoph Busch (3 shared papers)Anuradha Kaistha (3 shared papers)Han Si (2 shared papers)Rudolf Schubert (1 shared paper)
- Journals
- PLoS ONE (2 papers)Circulation (1 paper)European Journal of Cell Biology (1 paper)Arteriosclerosis Thrombosis and Vascular Biology (1 paper)Kidney & Blood Pressure Research (1 paper)
- Partner nations
- GermanyUnited StatesSpain
In The Last Decade
Michael Kacik
6 papers receiving 785 citations
Peers
Comparison fields: 5 of 70
- Sensory Systems 380
- Physiology 351
- Biochemistry 60
- Cardiology and Cardiovascular Medicine 188
- Endocrine and Autonomic Systems 47
Countries citing papers authored by Michael Kacik
This map shows the geographic impact of Michael Kacik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Kacik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Kacik more than expected).
Fields of papers citing papers by Michael Kacik
This network shows the impact of papers produced by Michael Kacik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Kacik. The network helps show where Michael Kacik may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael Kacik, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 279 | |
| 2 | 2007 | 232 | |
| 3 | 2009 | 210 | |
| 4 | 2015 | 52 | |
| 5 | 2014 | 10 | |
| 6 | 2019 | 6 |
About Michael Kacik
Michael Kacik is a scholar working on Sensory Systems, Physiology, Molecular Biology, Biochemistry and Surgery, having authored 6 papers that have together received 789 indexed citations. Recurring topics across this work include Nitric Oxide and Endothelin Effects (3 papers), Ion Channels and Receptors (3 papers), Eicosanoids and Hypertension Pharmacology (2 papers), Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper), MicroRNA in disease regulation (1 paper), Heart Rate Variability and Autonomic Control (1 paper), Biochemical Analysis and Sensing Techniques (1 paper) and Ion Transport and Channel Regulation (1 paper). The work is most often cited by research in Sensory Systems (380 citations), Physiology (351 citations), Biochemistry (60 citations), Cardiology and Cardiovascular Medicine (188 citations) and Endocrine and Autonomic Systems (47 citations). Michael Kacik has collaborated with scholars based in Germany, United States and Spain. Frequent co-authors include Joachim Hoyer, Ivica Grgic, Ralf Köhler, Willm‐Thomas Heyken, Christoph Busch, Anuradha Kaistha, Han Si, Rudolf Schubert, Tanja Maier and Christian Harteneck. Their work appears in journals such as PLoS ONE, Circulation, European Journal of Cell Biology, Arteriosclerosis Thrombosis and Vascular Biology and Kidney & Blood Pressure Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.