Michael Eichenlaub
Impact in
- Aging top 10%
-
- Genomics and Chromatin Dynamics
- Developmental Biology and Gene Regulation
- RNA Research and Splicing
- Pluripotent Stem Cells Research
- CRISPR and Genetic Engineering
- Muscle Physiology and Disorders
Papers in
-
- Genomics and Chromatin Dynamics 4
- Pluripotent Stem Cells Research 4
- CRISPR and Genetic Engineering 4
- Congenital heart defects research 3
- Developmental Biology and Gene Regulation 3
- Retinal Development and Disorders 2
- Genetics 5
- Animal Genetics and Reproduction 4
- Co-authors
- Eileen E. M. Furlong (2 shared papers)Thomas Sandmann (2 shared papers)Janus S. Jakobsen (1 shared paper)Peer Bork (1 shared paper)Lars Juhl Jensen (1 shared paper)Laurence Ettwiller (1 shared paper)David N. Wells (3 shared papers)Björn Oback (3 shared papers)
- Journals
- Biology of Reproduction (2 papers)Differentiation (1 paper)Reproduction Fertility and Development (1 paper)PLoS Biology (1 paper)Mutation Research/Genetic Toxicology and Environmental Mutagenesis (1 paper)
- Partner nations
- GermanyAustraliaUnited States
In The Last Decade
Michael Eichenlaub
14 papers receiving 462 citations
Peers
Comparison fields: 5 of 56
- Aging 23
- Molecular Biology 410
- Reproductive Medicine 41
- Genetics 106
- Public Health, Environmental and Occupational Health 80
Countries citing papers authored by Michael Eichenlaub
This map shows the geographic impact of Michael Eichenlaub's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Eichenlaub with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Eichenlaub more than expected).
Fields of papers citing papers by Michael Eichenlaub
This network shows the impact of papers produced by Michael Eichenlaub. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Eichenlaub. The network helps show where Michael Eichenlaub may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael Eichenlaub, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 198 | |
| 2 | 2017 | 54 | |
| 3 | 2006 | 50 | |
| 4 | 2011 | 48 | |
| 5 | 2010 | 28 | |
| 6 | 2016 | 25 | |
| 7 | 2016 | 23 | |
| 8 | 2016 | 17 | |
| 9 | 2012 | 10 | |
| 10 | 2021 | 7 | |
| 11 | 2020 | 5 | |
| 12 | 2025 | 2 | |
| 13 | 2021 | 1 | |
| 14 | 2012 | 1 | |
| 15 | 2024 | 0 |
About Michael Eichenlaub
Michael Eichenlaub is a scholar working on Molecular Biology, Genetics, Public Health, Environmental and Occupational Health, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine, having authored 15 papers that have together received 469 indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (4 papers), Pluripotent Stem Cells Research (4 papers), CRISPR and Genetic Engineering (4 papers), Animal Genetics and Reproduction (4 papers), Reproductive Biology and Fertility (3 papers), Congenital heart defects research (3 papers), Developmental Biology and Gene Regulation (3 papers) and Retinal Development and Disorders (2 papers). The work is most often cited by research in Aging (23 citations), Molecular Biology (410 citations), Reproductive Medicine (41 citations), Genetics (106 citations) and Public Health, Environmental and Occupational Health (80 citations). Michael Eichenlaub has collaborated with scholars based in Germany, Australia and United States. Frequent co-authors include Eileen E. M. Furlong, Thomas Sandmann, Janus S. Jakobsen, Peer Bork, Lars Juhl Jensen, Laurence Ettwiller, David N. Wells, Björn Oback, Mirana Ramialison and Robin M. Hobbs. Their work appears in journals such as Biology of Reproduction, Differentiation, Reproduction Fertility and Development, PLoS Biology and Mutation Research/Genetic Toxicology and Environmental Mutagenesis.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.