Michael E. Fealey

586 citations
22 papers · 405 · h-index 10

Impact in

Papers in

    • Lipid Membrane Structure and Behavior 5
    • Protein Structure and Dynamics 2
    • Cellular transport and secretion 5
    • Cellular Mechanics and Interactions 2

Michael E. Fealey

20 papers receiving 392 citations

Peers

Michael E. Fealey
Comparison fields: 5 of 57
  • Cardiology and Cardiovascular Medicine 131
  • Internal Medicine 14
  • Genetics 41
  • Endocrinology, Diabetes and Metabolism 51
  • Cell Biology 53
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Citations per year

Countries citing papers authored by Michael E. Fealey

Since Specialization
Citations

This map shows the geographic impact of Michael E. Fealey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael E. Fealey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael E. Fealey more than expected).

Fields of papers citing papers by Michael E. Fealey

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael E. Fealey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael E. Fealey. The network helps show where Michael E. Fealey may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael E. Fealey, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael E. Fealey Line = papers co-authored together Michael E. Fealey links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 22 papers — load more, or switch the sort, to bring in the rest.

#Work
1 200875
2 200765
3 201146
4 200941
5 201739
6 200829
7 201015
8 201814
9 201114
10 201211
11 20229
12 20189
13 20168
14 20087
15 20076
16 20145
17 20134
18 20064
19 20093
20 20091

About Michael E. Fealey

Michael E. Fealey is a scholar working on Molecular Biology, Cell Biology, Physiology, Cardiology and Cardiovascular Medicine and Epidemiology, having authored 22 papers that have together received 405 indexed citations. Recurring topics across this work include Cellular transport and secretion (5 papers), Lipid Membrane Structure and Behavior (5 papers), Erythrocyte Function and Pathophysiology (3 papers), Aortic Disease and Treatment Approaches (2 papers), Cardiac tumors and thrombi (2 papers), Protein Structure and Dynamics (2 papers), Advanced Fluorescence Microscopy Techniques (2 papers) and Cellular Mechanics and Interactions (2 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (131 citations), Internal Medicine (14 citations), Genetics (41 citations), Endocrinology, Diabetes and Metabolism (51 citations) and Cell Biology (53 citations). Michael E. Fealey has collaborated with scholars based in United States and Canada. Frequent co-authors include William D. Edwards, Joseph A. Dearani, Charanjit S. Rihal, Caterina Giannini, Harry J. Cloft, Carol Reynolds, Patricia A. Pellikka, David G. Piepgras, Anne Hinderliter and David D. Thomas. Their work appears in journals such as Cardiovascular Pathology, Biophysical Journal, Journal of Cardiology, Endocrine Pathology and Journal of Neuro-Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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