Michael E. Bayer
Impact in
- Cell Biology top 5%
- Cellular transport and secretion
- Hippo pathway signaling and YAP/TAZ
- Endoplasmic Reticulum Stress and Disease
- Nephrology top 10%
- Renal Diseases and Glomerulopathies
Papers in
-
- Cellular transport and secretion 3
-
- Immunotherapy and Immune Responses 3
- Co-authors
- Thomas Weide (4 shared papers)Angelika Barnekow (3 shared papers)Reiner Peters (1 shared paper)Jan Peter Siebrasse (1 shared paper)Albrecht Schwab (2 shared papers)Tobias Bruno Huber (2 shared papers)Thomas R. Benzing (2 shared papers)Joachim Kremerskothen (2 shared papers)
- Journals
- Journal for ImmunoTherapy of Cancer (2 papers)Blood (1 paper)American Journal Of Pathology (1 paper)Recent results in cancer research (1 paper)BMC Cell Biology (1 paper)
- Partner nations
- GermanyChinaNetherlands
In The Last Decade
Michael E. Bayer
11 papers receiving 474 citations
Peers
Comparison fields: 5 of 51
- Cell Biology 224
- Nephrology 54
- Hematology 35
- Cellular and Molecular Neuroscience 28
- Endocrinology 7
Countries citing papers authored by Michael E. Bayer
This map shows the geographic impact of Michael E. Bayer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael E. Bayer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael E. Bayer more than expected).
Fields of papers citing papers by Michael E. Bayer
This network shows the impact of papers produced by Michael E. Bayer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael E. Bayer. The network helps show where Michael E. Bayer may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael E. Bayer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2001 | 109 | |
| 2 | 2008 | 108 | |
| 3 | 2006 | 57 | |
| 4 | 2003 | 55 | |
| 5 | 2012 | 55 | |
| 6 | 2015 | 31 | |
| 7 | 2005 | 25 | |
| 8 | 2009 | 18 | |
| 9 | 2008 | 16 | |
| 10 | 2019 | 5 | |
| 11 | 2014 | 1 | |
| 12 | 2014 | 0 |
About Michael E. Bayer
Michael E. Bayer is a scholar working on Cell Biology, Immunology, Nephrology, Urology and Hematology, having authored 12 papers that have together received 480 indexed citations. Recurring topics across this work include Cellular transport and secretion (3 papers), Immunotherapy and Immune Responses (3 papers), Renal Diseases and Glomerulopathies (2 papers), Angiogenesis and VEGF in Cancer (2 papers), Acute Myeloid Leukemia Research (1 paper), Hair Growth and Disorders (1 paper) and Bone and Dental Protein Studies (1 paper). The work is most often cited by research in Cell Biology (224 citations), Nephrology (54 citations), Hematology (35 citations), Cellular and Molecular Neuroscience (28 citations) and Endocrinology (7 citations). Michael E. Bayer has collaborated with scholars based in Germany, China and Netherlands. Frequent co-authors include Thomas Weide, Angelika Barnekow, Reiner Peters, Jan Peter Siebrasse, Albrecht Schwab, Tobias Bruno Huber, Thomas R. Benzing, Joachim Kremerskothen, Torsten Keßler and Wolfgang E. Berdel. Their work appears in journals such as Journal for ImmunoTherapy of Cancer, Blood, American Journal Of Pathology, Recent results in cancer research and BMC Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.