Michael D. Bright

936 citations
17 papers · 755 · h-index 12

Impact in

Papers in

    • Endoplasmic Reticulum Stress and Disease 4
    • Protein Degradation and Inhibitors 5
    • Histone Deacetylase Inhibitors Research 3
    • Protein Kinase Regulation and GTPase Signaling 2
    • Epigenetics and DNA Methylation 2

Michael D. Bright

17 papers receiving 744 citations

Peers

Michael D. Bright
Comparison fields: 5 of 78
  • Endocrinology 229
  • Cell Biology 131
  • Infectious Diseases 130
  • Molecular Medicine 35
  • Hematology 65
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Jixin Cui United States
Aleksandra Bankovacki Australia
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Karel H. M. van Wely Spain
Irina I. Mirkina Austria
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Citations per field
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Citations per year

Countries citing papers authored by Michael D. Bright

Since Specialization
Citations

This map shows the geographic impact of Michael D. Bright's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael D. Bright with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael D. Bright more than expected).

Fields of papers citing papers by Michael D. Bright

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael D. Bright. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael D. Bright. The network helps show where Michael D. Bright may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael D. Bright, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael D. Bright Line = papers co-authored together Michael D. Bright links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 2011298
2 201793
3 202060
4 201549
5 201847
6 201241
7 200935
8 201829
9 201822
10 202219
11 201418
12 202112
13 201111
14 202211
15 20237
16 20162
17 20141

About Michael D. Bright

Michael D. Bright is a scholar working on Cell Biology, Molecular Biology, Oncology, Radiology, Nuclear Medicine and Imaging and Endocrinology, having authored 17 papers that have together received 755 indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (5 papers), Endoplasmic Reticulum Stress and Disease (4 papers), CAR-T cell therapy research (3 papers), Histone Deacetylase Inhibitors Research (3 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), Epigenetics and DNA Methylation (2 papers) and Autophagy in Disease and Therapy (2 papers). The work is most often cited by research in Endocrinology (229 citations), Cell Biology (131 citations), Infectious Diseases (130 citations), Molecular Medicine (35 citations) and Hematology (65 citations). Michael D. Bright has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include Gad M. Frankel, Diana Munera, Keith S. Robinson, Sau Fung Lee, Elizabeth Louise Hartland, Jaclyn S. Pearson, Faith E. Davies, Gareth John Morgan, Paul Workman and Anne J. Ridley. Their work appears in journals such as Journal of Medicinal Chemistry, Cellular Signalling, Blood, The International Journal of Biochemistry & Cell Biology and Journal of Cell Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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