Michael B. Dilling

1.3k citations
8 papers · 1.2k · h-index 8

Impact in

    • PI3K/AKT/mTOR signaling in cancer
    • Protein Kinase Regulation and GTPase Signaling
    • Polyamine Metabolism and Applications
    • Ubiquitin and proteasome pathways
    • Cell death mechanisms and regulation
  • Oncology top 10%
    • Cancer-related Molecular Pathways

Papers in

    • PI3K/AKT/mTOR signaling in cancer 5
    • Cancer therapeutics and mechanisms 2
    • DNA Repair Mechanisms 1
    • Ubiquitin and proteasome pathways 1
    • Melanoma and MAPK Pathways 1
    • Cell death mechanisms and regulation 1
    • Peptidase Inhibition and Analysis 1

Michael B. Dilling

8 papers receiving 1.1k citations

Peers

Michael B. Dilling
Comparison fields: 5 of 70
  • Molecular Biology 850
  • Oncology 279
  • Cancer Research 132
  • Genetics 81
  • Pathology and Forensic Medicine 134
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Citations per field
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Citations per year

Countries citing papers authored by Michael B. Dilling

Since Specialization
Citations

This map shows the geographic impact of Michael B. Dilling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael B. Dilling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael B. Dilling more than expected).

Fields of papers citing papers by Michael B. Dilling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael B. Dilling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael B. Dilling. The network helps show where Michael B. Dilling may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael B. Dilling, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael B. Dilling Line = papers co-authored together Michael B. Dilling links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1
Rapamycin causes poorly reversible inhibition of mTOR and induces p53-independent apoptosis in human rhabdomyosarcoma cells.
1999267
2
Biochemical correlates of mTOR inhibition by the rapamycin ester CCI-779 and tumor growth inhibition.
2001205
3 2003195
4 1998160
5
Rapamycin selectively inhibits the growth of childhood rhabdomyosarcoma cells through inhibition of signaling via the type I insulin-like growth factor receptor.
1994118
6
p53/p21(CIP1) cooperate in enforcing rapamycin-induced G(1) arrest and determine the cellular response to rapamycin.
2001114
7 200290
8 200011

About Michael B. Dilling

Michael B. Dilling is a scholar working on Molecular Biology, Oncology, Toxicology, Endocrinology, Diabetes and Metabolism and Biotechnology, having authored 8 papers that have together received 1.2k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (5 papers), Bioactive Compounds and Antitumor Agents (2 papers), Cancer therapeutics and mechanisms (2 papers), DNA Repair Mechanisms (1 paper), Ubiquitin and proteasome pathways (1 paper), Melanoma and MAPK Pathways (1 paper), Cell death mechanisms and regulation (1 paper) and Peptidase Inhibition and Analysis (1 paper). The work is most often cited by research in Molecular Biology (850 citations), Oncology (279 citations), Cancer Research (132 citations), Genetics (81 citations) and Pathology and Forensic Medicine (134 citations). Michael B. Dilling has collaborated with scholars based in United States, Netherlands and Japan. Frequent co-authors include Peter J. Houghton, Hajime Hosoi, Takuma Shikata, Linda N. Liu, Glen S. Germain, Franklin C. Harwood, Lili Shu, Peter J. Houghton, Robert T. Abraham and Shile Huang. Their work appears in journals such as Cancer Chemotherapy and Pharmacology, Journal of Biological Chemistry, Molecular Cell, Molecular Pharmacology and PubMed.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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