Max Costa
Impact in
-
- Heavy Metal Exposure and Toxicity
- Chromium effects and bioremediation
- Environmental Toxicology and Ecotoxicology
- Cancer Research top 10%
- Carcinogens and Genotoxicity Assessment
Papers in
-
- Genomics and Chromatin Dynamics 2
- DNA and Nucleic Acid Chemistry 2
- Molecular Biology Techniques and Applications 2
-
- Trace Elements in Health 4
- Co-authors
- Pramila Sen (1 shared paper)Orazio Cantoni (2 shared papers)Konstantin Salnikow (1 shared paper)Steven H. Robison (1 shared paper)Yong‐Woo Lee (1 shared paper)Limor Broday (1 shared paper)Jingxia Li (2 shared papers)Chuanshu Huang (2 shared papers)
- Journals
- Carcinogenesis (4 papers)Cancer Research (1 paper)Cell Cycle (1 paper)Mutation Research/Genetic Toxicology and Environmental Mutagenesis (1 paper)Biochemistry (1 paper)
- Partner nations
- United StatesBrazilChina
In The Last Decade
Max Costa
15 papers receiving 692 citations
Peers
Comparison fields: 5 of 90
- Health, Toxicology and Mutagenesis 248
- Cancer Research 218
- Nutrition and Dietetics 165
- Molecular Biology 332
- Pollution 48
Countries citing papers authored by Max Costa
This map shows the geographic impact of Max Costa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Max Costa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Max Costa more than expected).
Fields of papers citing papers by Max Costa
This network shows the impact of papers produced by Max Costa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Max Costa. The network helps show where Max Costa may publish in the future.
Co-authors
The 25 scholars most cited alongside Max Costa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Induction of chromosomal damage in Chinese hamster ovary cells by soluble and particulate nickel compounds: preferential fragmentation of the heterochromatic long arm of the X-chromosome by carcinogenic crystalline NiS particles. | 1985 | 105 |
| 2 | 2010 | 88 | |
| 3 | 1982 | 82 | |
| 4 | 2009 | 76 | |
| 5 | 1998 | 74 | |
| 6 | Epigenetic mechanisms of nickel carcinogenesis. | 2000 | 61 |
| 7 | Preferential DNA-protein cross-linking by NiCl2 in magnesium-insoluble regions of fractionated Chinese hamster ovary cell chromatin. | 1985 | 55 |
| 8 | 1987 | 53 | |
| 9 | 2009 | 52 | |
| 10 | 1984 | 37 | |
| 11 | 2005 | 13 | |
| 12 | 2013 | 9 | |
| 13 | 1991 | 7 | |
| 14 | 2015 | 6 | |
| 15 | Mechanisms of stability and reversion of mass conversion stable l forms of bacillus subtilis | 1977 | 3 |
About Max Costa
Max Costa is a scholar working on Molecular Biology, Nutrition and Dietetics, Cancer Research, Physiology and Inorganic Chemistry, having authored 15 papers that have together received 721 indexed citations. Recurring topics across this work include Trace Elements in Health (4 papers), Genomics and Chromatin Dynamics (2 papers), DNA and Nucleic Acid Chemistry (2 papers), Vanadium and Halogenation Chemistry (2 papers), Molecular Biology Techniques and Applications (2 papers), Carcinogens and Genotoxicity Assessment (1 paper), MicroRNA in disease regulation (1 paper) and Potato Plant Research (1 paper). The work is most often cited by research in Health, Toxicology and Mutagenesis (248 citations), Cancer Research (218 citations), Nutrition and Dietetics (165 citations), Molecular Biology (332 citations) and Pollution (48 citations). Max Costa has collaborated with scholars based in United States, Brazil and China. Frequent co-authors include Pramila Sen, Orazio Cantoni, Konstantin Salnikow, Steven H. Robison, Yong‐Woo Lee, Limor Broday, Jingxia Li, Chuanshu Huang, Dongyun Zhang and Jimin Gao. Their work appears in journals such as Carcinogenesis, Cancer Research, Cell Cycle, Mutation Research/Genetic Toxicology and Environmental Mutagenesis and Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.