Matthew E. Burow

18.0k citations
204 papers · 9.4k · h-index 54

Impact in

    • MicroRNA in disease regulation
    • Cancer-related molecular mechanisms research
  • Oncology top 1%
    • Cancer Cells and Metastasis

Papers in

    • Melanoma and MAPK Pathways 17
    • Sphingolipid Metabolism and Signaling 11
    • Cancer Cells and Metastasis 37
    • Cytokine Signaling Pathways and Interactions 11

Matthew E. Burow

195 papers receiving 9.1k citations

Peers

Matthew E. Burow
Comparison fields: 5 of 155
  • Cancer Research 1.7k
  • Oncology 2.0k
  • Molecular Biology 4.4k
  • Biochemistry 374
  • Pathology and Forensic Medicine 882
Replace Janusz Błasiak with:
Janusz Błasiak Poland
Sebastiano Andò Italy
Alicja Józkowicz Poland
Matthew R. Young United States
Kyung‐Chul Choi South Korea
Judith Haendeler Germany
Hung‐Yun Lin United States
Shuyuan Yeh United States
Toshio Kuroki Japan
Kyung‐Sun Kang South Korea
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Citations per field
00.5×1.5×2.1×
Janusz Błasiak · 1×
Citations per year

Countries citing papers authored by Matthew E. Burow

Since Specialization
Citations

This map shows the geographic impact of Matthew E. Burow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew E. Burow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew E. Burow more than expected).

Fields of papers citing papers by Matthew E. Burow

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew E. Burow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew E. Burow. The network helps show where Matthew E. Burow may publish in the future.

Co-authors

The 25 scholars most cited alongside Matthew E. Burow, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Matthew E. Burow Line = papers co-authored together Matthew E. Burow links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 204 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2000374
2 2010299
3 2003276
4 2007262
5 2003228
6 2012224
7 2004221
8 2003204
9 2017197
10
Differences in susceptibility to tumor necrosis factor alpha-induced apoptosis among MCF-7 breast cancer cell variants.
1998167
11 2015156
12 2011152
13 2010147
14 2013133
15 2000131
16 2012130
17 2011129
18 2010125
19 2014111
20 2006109

About Matthew E. Burow

Matthew E. Burow is a scholar working on Molecular Biology, Oncology, Cancer Research, Genetics and Pathology and Forensic Medicine, having authored 204 papers that have together received 9.4k indexed citations. Recurring topics across this work include Cancer Cells and Metastasis (37 papers), Estrogen and related hormone effects (30 papers), Phytoestrogen effects and research (22 papers), Melanoma and MAPK Pathways (17 papers), MicroRNA in disease regulation (14 papers), Effects and risks of endocrine disrupting chemicals (12 papers), Sphingolipid Metabolism and Signaling (11 papers) and Cytokine Signaling Pathways and Interactions (11 papers). The work is most often cited by research in Cancer Research (1.7k citations), Oncology (2.0k citations), Molecular Biology (4.4k citations), Biochemistry (374 citations) and Pathology and Forensic Medicine (882 citations). Matthew E. Burow has collaborated with scholars based in United States, China and United Kingdom. Frequent co-authors include John A. McLachlan, Barbara S. Beckman, Bridgette M. Collins‐Burow, Steven Elliott, Lyndsay V. Rhodes, Christopher B. Weldon, Bruce A. Bunnell, Stephen M. Boué, Amy L. Strong and James W. Antoon. Their work appears in journals such as Breast Cancer Research, PLoS ONE, Journal of Agricultural and Food Chemistry, Cancer Research and Molecular Carcinogenesis.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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