Mathew Cox

4.5k citations
6 papers · 394 · h-index 5

Impact in

    • MicroRNA in disease regulation
    • Cancer-related molecular mechanisms research
    • Pregnancy and preeclampsia studies
    • Gestational Diabetes Research and Management

Papers in

Mathew Cox

6 papers receiving 391 citations

Peers

Mathew Cox
Comparison fields: 5 of 54
  • Cancer Research 170
  • Obstetrics and Gynecology 66
  • Pathology and Forensic Medicine 92
  • Immunology 85
  • Pediatrics, Perinatology and Child Health 72
Replace Olga Kovalenko with:
Olga Kovalenko United States
Xiao-Ying Hao United States
Hadas Grossman Israel
Sheena L. M. Ong Singapore
Yuanhui Jia China
Erina Suzuki Japan
Adrienne S. McCampbell United States
Amita Limaye India
Luca Mariani Italy
Kristofer C. Berrett United States
Mathew Cox relative to Olga Kovalenko United States Olga Kovalenko's profile →
Citations per field
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Citations per year

Countries citing papers authored by Mathew Cox

Since Specialization
Citations

This map shows the geographic impact of Mathew Cox's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathew Cox with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathew Cox more than expected).

Fields of papers citing papers by Mathew Cox

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathew Cox. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathew Cox. The network helps show where Mathew Cox may publish in the future.

Co-authors

The 23 scholars most cited alongside Mathew Cox, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mathew Cox Line = papers co-authored together Mathew Cox links everyone, so they are left out of the graph.

All Works

6 of 6 papers shown
#Work
1 2010196
2 201293
3 201148
4 201046
5 20138
6 20133

About Mathew Cox

Mathew Cox is a scholar working on Immunology, Molecular Biology, Cancer Research, Pathology and Forensic Medicine and Pediatrics, Perinatology and Child Health, having authored 6 papers that have together received 394 indexed citations. Recurring topics across this work include Galectins and Cancer Biology (2 papers), Protease and Inhibitor Mechanisms (2 papers), MicroRNA in disease regulation (1 paper), T-cell and Retrovirus Studies (1 paper), Cell Adhesion Molecules Research (1 paper), High Altitude and Hypoxia (1 paper), Whipple's Disease and Interleukins (1 paper) and Psoriasis: Treatment and Pathogenesis (1 paper). The work is most often cited by research in Cancer Research (170 citations), Obstetrics and Gynecology (66 citations), Pathology and Forensic Medicine (92 citations), Immunology (85 citations) and Pediatrics, Perinatology and Child Health (72 citations). Mathew Cox has collaborated with scholars based in Australia, United Kingdom and Iran. Frequent co-authors include Jeannette Lechner‐Scott, Rodney J. Scott, Martha C. Tissot van Patot, Graham J. Burton, Hong Wa Yung, Graeme J. Stewart, Kaushal Gandhi, David R. Booth, Simon Broadley and Murray J. Cairns. Their work appears in journals such as Multiple Sclerosis Journal, PLoS ONE and The FASEB Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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