Marta Davidson

955 citations
21 papers · 653 · h-index 7

Impact in

    • DNA Repair Mechanisms
    • Genomics and Chromatin Dynamics
    • RNA Research and Splicing
    • Nuclear Structure and Function
    • CRISPR and Genetic Engineering
    • Ubiquitin and proteasome pathways
    • Fungal and yeast genetics research

Papers in

    • Acute Myeloid Leukemia Research 9
    • Chronic Myeloid Leukemia Treatments 6
    • Myeloproliferative Neoplasms: Diagnosis and Treatment 7

Marta Davidson

18 papers receiving 651 citations

Peers

Marta Davidson
Comparison fields: 5 of 71
  • Molecular Biology 571
  • Aging 7
  • Cell Biology 61
  • Cancer Research 44
  • Hematology 31
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Citations per field
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Citations per year

Countries citing papers authored by Marta Davidson

Since Specialization
Citations

This map shows the geographic impact of Marta Davidson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marta Davidson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marta Davidson more than expected).

Fields of papers citing papers by Marta Davidson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marta Davidson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marta Davidson. The network helps show where Marta Davidson may publish in the future.

Co-authors

The 25 scholars most cited alongside Marta Davidson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Marta Davidson Line = papers co-authored together Marta Davidson links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 21 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2008375
2 2012102
3 201285
4 201528
5 200817
6 202310
7 20248
8 20246
9 20216
10 20224
11 20093
12 20252
13 20222
14 20231
15 20171
16 20241
17 20221
18 20221
19 20210
20 20240

About Marta Davidson

Marta Davidson is a scholar working on Hematology, Genetics, Molecular Biology, Genetics and Pathology and Forensic Medicine, having authored 21 papers that have together received 653 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (9 papers), Myeloproliferative Neoplasms: Diagnosis and Treatment (7 papers), Chronic Myeloid Leukemia Treatments (6 papers), DNA Repair Mechanisms (5 papers), Cancer Genomics and Diagnostics (2 papers), Lymphoma Diagnosis and Treatment (2 papers), Genomics and Chromatin Dynamics (2 papers) and Epigenetics and DNA Methylation (1 paper). The work is most often cited by research in Molecular Biology (571 citations), Aging (7 citations), Cell Biology (61 citations), Cancer Research (44 citations) and Hematology (31 citations). Marta Davidson has collaborated with scholars based in Canada, United States and Australia. Frequent co-authors include Grant W. Brown, Susan M. Gasser, Florence Hediger, Monika Tsai-Pflugfelder, Shigeki Nagai, Elisa Varela, Karine Dubrana, Nevan J. Krogan, Tania Michelle Roberts and David Cawthorpe. Their work appears in journals such as Blood, Blood Advances, DNA repair, PLoS ONE and Molecular Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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