Markus Bach
Impact in
- Physiology top 5%
- Calcium signaling and nucleotide metabolism
- Epidemiology top 10%
- Autophagy in Disease and Therapy
Papers in
-
- Ubiquitin and proteasome pathways 1
- Mitochondrial Function and Pathology 1
- Oncology 3
- Drug Transport and Resistance Mechanisms 3
- Co-authors
- Georg Ramm (3 shared papers)Mark Larance (1 shared paper)David E. James (1 shared paper)Dirk Gründemann (5 shared papers)Edgar Schömig (4 shared papers)Benjamin Scott Padman (2 shared papers)Christian Fork (3 shared papers)Mark Prescott (1 shared paper)
- Journals
- Biochemical Journal (2 papers)FEBS Journal (1 paper)Drug Metabolism and Disposition (1 paper)Biochimica et Biophysica Acta (BBA) - Biomembranes (1 paper)Free Radical Biology and Medicine (1 paper)
- Partner nations
- GermanyAustraliaSwitzerland
In The Last Decade
Markus Bach
8 papers receiving 487 citations
Peers
Comparison fields: 5 of 74
- Physiology 51
- Epidemiology 218
- Clinical Biochemistry 46
- Biochemistry 41
- Pharmacology 43
Countries citing papers authored by Markus Bach
This map shows the geographic impact of Markus Bach's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Markus Bach with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Markus Bach more than expected).
Fields of papers citing papers by Markus Bach
This network shows the impact of papers produced by Markus Bach. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Markus Bach. The network helps show where Markus Bach may publish in the future.
Co-authors
The 16 scholars most cited alongside Markus Bach, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 213 | |
| 2 | 2013 | 81 | |
| 3 | 2008 | 71 | |
| 4 | 2015 | 46 | |
| 5 | 2007 | 40 | |
| 6 | 2009 | 25 | |
| 7 | 2014 | 15 | |
| 8 | 2020 | 4 |
About Markus Bach
Markus Bach is a scholar working on Molecular Biology, Oncology, Cellular and Molecular Neuroscience, Epidemiology and Pharmacology, having authored 8 papers that have together received 495 indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (3 papers), Pharmacogenetics and Drug Metabolism (2 papers), Amino Acid Enzymes and Metabolism (2 papers), Autophagy in Disease and Therapy (2 papers), Neuroscience and Neural Engineering (1 paper), Ubiquitin and proteasome pathways (1 paper), Alzheimer's disease research and treatments (1 paper) and Mitochondrial Function and Pathology (1 paper). The work is most often cited by research in Physiology (51 citations), Epidemiology (218 citations), Clinical Biochemistry (46 citations), Biochemistry (41 citations) and Pharmacology (43 citations). Markus Bach has collaborated with scholars based in Germany, Australia and Switzerland. Frequent co-authors include Georg Ramm, Mark Larance, David E. James, Dirk Gründemann, Edgar Schömig, Benjamin Scott Padman, Christian Fork, Mark Prescott, Stefan Gölz and Andreas Geerts. Their work appears in journals such as Biochemical Journal, FEBS Journal, Drug Metabolism and Disposition, Biochimica et Biophysica Acta (BBA) - Biomembranes and Free Radical Biology and Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.