Mark P. Hanson
Impact in
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- Genetic Neurodegenerative Diseases
- Neuroscience and Neuropharmacology Research
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- Cardiac electrophysiology and arrhythmias
Papers in
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- Neurofibromatosis and Schwannoma Cases 2
- Neuroblastoma Research and Treatments 1
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- Tumors and Oncological Cases 1
- Co-authors
- Bertrand Fontaine (4 shared papers)James F. Gusella (4 shared papers)Robert H. Brown (1 shared paper)Donna Romano (1 shared paper)G.A.P. Bruns (1 shared paper)Jonathan L. Haines (1 shared paper)James A. Trofatter (1 shared paper)Eric Hoffman (1 shared paper)
- Journals
- Annals of Neurology (1 paper)Genomics (1 paper)Science (1 paper)Journal of Bacteriology (1 paper)Microbiology (1 paper)
- Partner nations
- United StatesFranceCanada
In The Last Decade
Mark P. Hanson
6 papers receiving 322 citations
Peers
Comparison fields: 5 of 56
- Cellular and Molecular Neuroscience 142
- Cardiology and Cardiovascular Medicine 131
- Sensory Systems 21
- Molecular Biology 274
- Neurology 52
Countries citing papers authored by Mark P. Hanson
This map shows the geographic impact of Mark P. Hanson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark P. Hanson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark P. Hanson more than expected).
Fields of papers citing papers by Mark P. Hanson
This network shows the impact of papers produced by Mark P. Hanson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark P. Hanson. The network helps show where Mark P. Hanson may publish in the future.
Co-authors
The 24 scholars most cited alongside Mark P. Hanson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1990 | 266 | |
| 2 | 1993 | 21 | |
| 3 | 1991 | 19 | |
| 4 | 1991 | 18 | |
| 5 | 1997 | 14 | |
| 6 | Equal parental origin of chromosome 22 losses in human sporadic meningioma: no evidence for genomic imprinting. | 1990 | 11 |
About Mark P. Hanson
Mark P. Hanson is a scholar working on Neurology, Pathology and Forensic Medicine, Sensory Systems, Genetics and Molecular Biology, having authored 6 papers that have together received 349 indexed citations. Recurring topics across this work include Neurofibromatosis and Schwannoma Cases (2 papers), Microbial Metabolic Engineering and Bioproduction (1 paper), Enzyme Catalysis and Immobilization (1 paper), Bacterial Genetics and Biotechnology (1 paper), Genetic Syndromes and Imprinting (1 paper), Tumors and Oncological Cases (1 paper), Neuroblastoma Research and Treatments (1 paper) and Meningioma and schwannoma management (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (142 citations), Cardiology and Cardiovascular Medicine (131 citations), Sensory Systems (21 citations), Molecular Biology (274 citations) and Neurology (52 citations). Mark P. Hanson has collaborated with scholars based in United States, France and Canada. Frequent co-authors include Bertrand Fontaine, James F. Gusella, Robert H. Brown, Donna Romano, G.A.P. Bruns, Jonathan L. Haines, James A. Trofatter, Eric Hoffman, Tejvir S. Khurana and Carol A. Kumamoto. Their work appears in journals such as Annals of Neurology, Genomics, Science, Journal of Bacteriology and Microbiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.