Malcolm V. Gee

15 papers receiving 1.7k citations

Malcolm V. Gee's Hit Papers

NADPH cytochrome P-450 reductase activation of quinone anticancer agents to free radicals. 1979 · 453 citations
4530+16+32Years since publication200400600

Peers

Malcolm V. Gee
Comparison fields: 5 of 100
  • Toxicology 212
  • Biophysics 286
  • Cardiology and Cardiovascular Medicine 477
  • Oncology 513
  • Organic Chemistry 388
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Tomáš Šimůnek Czechia
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Huiying Li China
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Countries citing papers authored by Malcolm V. Gee

Since Specialization
Citations

This map shows the geographic impact of Malcolm V. Gee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Malcolm V. Gee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Malcolm V. Gee more than expected).

Fields of papers citing papers by Malcolm V. Gee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Malcolm V. Gee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Malcolm V. Gee. The network helps show where Malcolm V. Gee may publish in the future.

Co-authors

The 20 scholars most cited alongside Malcolm V. Gee, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Malcolm V. Gee Line = papers co-authored together Malcolm V. Gee links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1
A general mechanism for microsomal activation of quinone anticancer agents to free radicals.
Hit paper breakdown →
1978612
2
NADPH cytochrome P-450 reductase activation of quinone anticancer agents to free radicals.
Hit paper breakdown →
1979453
3 1977347
4
Nuclear catalyzed antibiotic free radical formation.
1982142
5 197495
6 197171
7 197644
8 198344
9 198841
10 198912
11 198612
12 19856
13 19662
14 19832
15 19691

About Malcolm V. Gee

Malcolm V. Gee is a scholar working on Molecular Biology, Toxicology, Biophysics, Physiology and Clinical Biochemistry, having authored 15 papers that have together received 1.9k indexed citations. Recurring topics across this work include Cancer therapeutics and mechanisms (4 papers), Receptor Mechanisms and Signaling (3 papers), Neuropeptides and Animal Physiology (3 papers), Chemotherapy-induced cardiotoxicity and mitigation (2 papers), Electron Spin Resonance Studies (2 papers), Metabolism and Genetic Disorders (2 papers), Bioactive Compounds and Antitumor Agents (2 papers) and Salivary Gland Disorders and Functions (2 papers). The work is most often cited by research in Toxicology (212 citations), Biophysics (286 citations), Cardiology and Cardiovascular Medicine (477 citations), Oncology (513 citations) and Organic Chemistry (388 citations). Malcolm V. Gee has collaborated with scholars based in United States. Frequent co-authors include Nicholas R. Bachur, Sandra Leigh Gordon, N R Bachur, Hirofumi Kon, S Gordon, Rosalind D. Friedman, Ronald L. Felsted, Bruce J. Baum, George S. Roth and G. S. Roth. Their work appears in journals such as Experimental Gerontology, Journal of Biological Chemistry, Journal of Pharmacology and Experimental Therapeutics, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research and Molecular Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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