Mailin Li
Impact in
-
- Cancer, Hypoxia, and Metabolism
-
- Immune cells in cancer
Papers in
-
- Heme Oxygenase-1 and Carbon Monoxide 6
- CRISPR and Genetic Engineering 1
- Epigenetics and DNA Methylation 1
-
- Cancer, Hypoxia, and Metabolism 4
- Co-authors
- Barbara Wegiel (9 shared papers)Eva Csizmadia (7 shared papers)Andreas Hedblom (4 shared papers)Pankaj Seth (2 shared papers)Han Xie (1 shared paper)Maria Serena Longhi (1 shared paper)Marta Vuerich (1 shared paper)Leo E. Otterbein (4 shared papers)
- Journals
- Clinical Cancer Research (2 papers)Oncotarget (2 papers)Cell Reports (1 paper)American Journal Of Pathology (1 paper)Bioorganic & Medicinal Chemistry Letters (1 paper)
- Partner nations
- United StatesHungaryAustralia
In The Last Decade
Mailin Li
11 papers receiving 345 citations
Peers
Comparison fields: 5 of 68
- Cancer Research 77
- Immunology 90
- Biochemistry 20
- Molecular Biology 186
- Biological Psychiatry 5
Countries citing papers authored by Mailin Li
This map shows the geographic impact of Mailin Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mailin Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mailin Li more than expected).
Fields of papers citing papers by Mailin Li
This network shows the impact of papers produced by Mailin Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mailin Li. The network helps show where Mailin Li may publish in the future.
Co-authors
The 25 scholars most cited alongside Mailin Li, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 121 | |
| 2 | 2016 | 46 | |
| 3 | 2015 | 46 | |
| 4 | 2020 | 38 | |
| 5 | 2016 | 26 | |
| 6 | 2014 | 18 | |
| 7 | 2019 | 16 | |
| 8 | 2016 | 15 | |
| 9 | 2017 | 13 | |
| 10 | 2020 | 6 | |
| 11 | 2023 | 2 | |
| 12 | 2025 | 0 | |
| 13 | 2026 | 0 |
About Mailin Li
Mailin Li is a scholar working on Molecular Biology, Cancer Research, Organic Chemistry, Pathology and Forensic Medicine and Pediatrics, Perinatology and Child Health, having authored 13 papers that have together received 347 indexed citations. Recurring topics across this work include Heme Oxygenase-1 and Carbon Monoxide (6 papers), Cancer, Hypoxia, and Metabolism (4 papers), Synthesis and biological activity (2 papers), Neonatal Health and Biochemistry (2 papers), Hemoglobin structure and function (2 papers), CRISPR and Genetic Engineering (1 paper), Eicosanoids and Hypertension Pharmacology (1 paper) and Epigenetics and DNA Methylation (1 paper). The work is most often cited by research in Cancer Research (77 citations), Immunology (90 citations), Biochemistry (20 citations), Molecular Biology (186 citations) and Biological Psychiatry (5 citations). Mailin Li has collaborated with scholars based in United States, Hungary and Australia. Frequent co-authors include Barbara Wegiel, Eva Csizmadia, Andreas Hedblom, Pankaj Seth, Han Xie, Maria Serena Longhi, Marta Vuerich, Leo E. Otterbein, Zsuzsanna Németh and Balázs Döme. Their work appears in journals such as Clinical Cancer Research, Oncotarget, Cell Reports, American Journal Of Pathology and Bioorganic & Medicinal Chemistry Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.