M. Funabiki
Impact in
- Immunology top 10%
- interferon and immune responses
- Immune Response and Inflammation
- Immune Cell Function and Interaction
Papers in
-
- Systemic Lupus Erythematosus Research 2
- Urologic and reproductive health conditions 1
-
- interferon and immune responses 3
- Immune Cell Function and Interaction 2
- Co-authors
- Hiroki Kato (4 shared papers)Takashi Fujita (4 shared papers)Katashi Deguchi (1 shared paper)Sadayoshi Ito (1 shared paper)Toshihiko Shiroishi (1 shared paper)Tatsuya Yamada (1 shared paper)Tetsuo Noda (1 shared paper)Hiroshi Satō (1 shared paper)
- Journals
- International Immunology (2 papers)The American Journal of Human Genetics (1 paper)Catalysis Today (1 paper)Clinical and Experimental Dermatology (1 paper)Immunity (1 paper)
- Partner nations
- JapanGermanyUnited States
In The Last Decade
M. Funabiki
8 papers receiving 448 citations
Peers
Comparison fields: 5 of 55
- Immunology 337
- Catalysis 41
- Rheumatology 82
- Molecular Biology 237
- Oncology 55
Countries citing papers authored by M. Funabiki
This map shows the geographic impact of M. Funabiki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Funabiki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Funabiki more than expected).
Fields of papers citing papers by M. Funabiki
This network shows the impact of papers produced by M. Funabiki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Funabiki. The network helps show where M. Funabiki may publish in the future.
Co-authors
The 25 scholars most cited alongside M. Funabiki, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2014 | 208 | |
| 2 | 2014 | 158 | |
| 3 | 1991 | 42 | |
| 4 | 2020 | 17 | |
| 5 | 1979 | 14 | |
| 6 | 2020 | 8 | |
| 7 | 2023 | 3 | |
| 8 | Advances in autoexhaust catalysts | 1997 | 2 |
| 9 | 2010 | 0 |
About M. Funabiki
M. Funabiki is a scholar working on Rheumatology, Immunology, Catalysis, Materials Chemistry and Surgery, having authored 9 papers that have together received 452 indexed citations. Recurring topics across this work include interferon and immune responses (3 papers), Catalytic Processes in Materials Science (3 papers), Catalysis and Oxidation Reactions (2 papers), Systemic Lupus Erythematosus Research (2 papers), Immune Cell Function and Interaction (2 papers), Gas Sensing Nanomaterials and Sensors (1 paper), Cytokine Signaling Pathways and Interactions (1 paper) and Urologic and reproductive health conditions (1 paper). The work is most often cited by research in Immunology (337 citations), Catalysis (41 citations), Rheumatology (82 citations), Molecular Biology (237 citations) and Oncology (55 citations). M. Funabiki has collaborated with scholars based in Japan, Germany and United States. Frequent co-authors include Hiroki Kato, Takashi Fujita, Katashi Deguchi, Sadayoshi Ito, Toshihiko Shiroishi, Tatsuya Yamada, Tetsuo Noda, Hiroshi Satō, Hiromi Motegi and Osamu Minowa. Their work appears in journals such as International Immunology, The American Journal of Human Genetics, Catalysis Today, Clinical and Experimental Dermatology and Immunity.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.