Loïc Fort
Impact in
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- Cellular Mechanics and Interactions
- Cellular transport and secretion
- Endoplasmic Reticulum Stress and Disease
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- Cell Adhesion Molecules Research
Papers in
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- Endoplasmic Reticulum Stress and Disease 2
- Cellular transport and secretion 2
- Cellular Mechanics and Interactions 2
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- Ubiquitin and proteasome pathways 1
- Co-authors
- Laura M. Machesky (7 shared papers)Nikki R. Paul (4 shared papers)Heather J. Spence (4 shared papers)Vivian Gama (1 shared paper)Shehab Ismail (1 shared paper)Ian G. Macara (1 shared paper)Sérgio Lilla (4 shared papers)Tamas Yelland (1 shared paper)
- Journals
- The Journal of Cell Biology (1 paper)Development (1 paper)Nature Cell Biology (1 paper)Communicative & Integrative Biology (1 paper)The Journal of Pathology (1 paper)
- Partner nations
- United KingdomUnited StatesSouth Sudan
In The Last Decade
Loïc Fort
9 papers receiving 172 citations
Peers
Comparison fields: 5 of 47
- Cell Biology 64
- Immunology and Allergy 19
- Aging 3
- Molecular Biology 97
- Immunology 25
Countries citing papers authored by Loïc Fort
This map shows the geographic impact of Loïc Fort's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Loïc Fort with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Loïc Fort more than expected).
Fields of papers citing papers by Loïc Fort
This network shows the impact of papers produced by Loïc Fort. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Loïc Fort. The network helps show where Loïc Fort may publish in the future.
Co-authors
The 25 scholars most cited alongside Loïc Fort, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 41 | |
| 2 | 2016 | 38 | |
| 3 | 2013 | 29 | |
| 4 | 2021 | 25 | |
| 5 | 2018 | 13 | |
| 6 | 2022 | 11 | |
| 7 | 2019 | 8 | |
| 8 | 2024 | 6 | |
| 9 | 2018 | 1 | |
| 10 | 2023 | 1 | |
| 11 | 2025 | 0 |
About Loïc Fort
Loïc Fort is a scholar working on Cell Biology, Molecular Biology, Immunology, Physiology and Neurology, having authored 11 papers that have together received 173 indexed citations. Recurring topics across this work include Phagocytosis and Immune Regulation (3 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Cellular transport and secretion (2 papers), Erythrocyte Function and Pathophysiology (2 papers), Cellular Mechanics and Interactions (2 papers), Amyotrophic Lateral Sclerosis Research (1 paper), Ubiquitin and proteasome pathways (1 paper) and Science, Research, and Medicine (1 paper). The work is most often cited by research in Cell Biology (64 citations), Immunology and Allergy (19 citations), Aging (3 citations), Molecular Biology (97 citations) and Immunology (25 citations). Loïc Fort has collaborated with scholars based in United Kingdom, United States and South Sudan. Frequent co-authors include Laura M. Machesky, Nikki R. Paul, Heather J. Spence, Vivian Gama, Shehab Ismail, Ian G. Macara, Sérgio Lilla, Tamas Yelland, Amélie Juin and Xiaocui Zhang. Their work appears in journals such as The Journal of Cell Biology, Development, Nature Cell Biology, Communicative & Integrative Biology and The Journal of Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.