Libin Wei
Impact in
- Cancer Research top 5%
- Cancer, Hypoxia, and Metabolism
- Cancer, Lipids, and Metabolism
- Pharmacology top 5%
- Flavonoids in Medical Research
Papers in
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- Cell death mechanisms and regulation 5
- Metabolism, Diabetes, and Cancer 4
- Ubiquitin and proteasome pathways 4
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- Cancer, Hypoxia, and Metabolism 16
- Cancer, Lipids, and Metabolism 7
- Co-authors
- Qinglong Guo (32 shared papers)Na Lu (23 shared papers)Zhiyu Li (18 shared papers)Jingyue Yao (5 shared papers)Qinsheng Dai (12 shared papers)Yongjian Guo (11 shared papers)Yuxin Zhou (9 shared papers)Ye Xu (3 shared papers)
- Journals
- Molecular Carcinogenesis (4 papers)Free Radical Biology and Medicine (3 papers)Oncotarget (3 papers)Cell Death and Disease (3 papers)Biomedicine & Pharmacotherapy (2 papers)
- Partner nations
- ChinaUnited StatesNorway
In The Last Decade
Libin Wei
47 papers receiving 1.4k citations
Libin Wei's Hit Papers
Peers
Comparison fields: 5 of 97
- Cancer Research 354
- Pharmacology 333
- Geriatrics and Gerontology 61
- Molecular Biology 868
- Physiology 35
Countries citing papers authored by Libin Wei
This map shows the geographic impact of Libin Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Libin Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Libin Wei more than expected).
Fields of papers citing papers by Libin Wei
This network shows the impact of papers produced by Libin Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Libin Wei. The network helps show where Libin Wei may publish in the future.
Co-authors
The 25 scholars most cited alongside Libin Wei, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 48 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | CDK9 inhibition blocks the initiation of PINK1-PRKN-mediated mitophagy by regulating the SIRT1-FOXO3-BNIP3 axis and enhances the therapeutic effects involving mitochondrial dysfunction in hepatocellular carcinoma Hit paper breakdown → | 2021 | 155 |
| 2 | 2021 | 96 | |
| 3 | 2017 | 74 | |
| 4 | 2013 | 66 | |
| 5 | 2015 | 55 | |
| 6 | 2019 | 46 | |
| 7 | 2018 | 43 | |
| 8 | 2015 | 42 | |
| 9 | 2021 | 41 | |
| 10 | 2013 | 40 | |
| 11 | 2014 | 40 | |
| 12 | 2014 | 39 | |
| 13 | 2011 | 36 | |
| 14 | 2010 | 36 | |
| 15 | 2015 | 35 | |
| 16 | 2015 | 35 | |
| 17 | 2013 | 33 | |
| 18 | 2016 | 32 | |
| 19 | 2015 | 32 | |
| 20 | 2014 | 32 |
About Libin Wei
Libin Wei is a scholar working on Molecular Biology, Cancer Research, Pharmacology, Oncology and Cell Biology, having authored 48 papers that have together received 1.4k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (16 papers), Flavonoids in Medical Research (15 papers), Cancer-related Molecular Pathways (8 papers), Cancer, Lipids, and Metabolism (7 papers), Cell death mechanisms and regulation (5 papers), Autophagy in Disease and Therapy (4 papers), Metabolism, Diabetes, and Cancer (4 papers) and Ubiquitin and proteasome pathways (4 papers). The work is most often cited by research in Cancer Research (354 citations), Pharmacology (333 citations), Geriatrics and Gerontology (61 citations), Molecular Biology (868 citations) and Physiology (35 citations). Libin Wei has collaborated with scholars based in China, United States and Norway. Frequent co-authors include Qinglong Guo, Na Lu, Zhiyu Li, Jingyue Yao, Qinsheng Dai, Yongjian Guo, Yuxin Zhou, Ye Xu, Qidong You and Yuxin Zhou. Their work appears in journals such as Molecular Carcinogenesis, Free Radical Biology and Medicine, Oncotarget, Cell Death and Disease and Biomedicine & Pharmacotherapy.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.