Kuan Ning
Impact in
- Cancer Research top 10%
- MicroRNA in disease regulation
- Cancer, Hypoxia, and Metabolism
- Cancer-related molecular mechanisms research
- Sensory Systems top 10%
- Ion Channels and Receptors
Papers in
-
- Receptor Mechanisms and Signaling 2
- Extracellular vesicles in disease 2
- DNA Repair Mechanisms 1
-
- Neuropeptides and Animal Physiology 2
- Co-authors
- Jian Jin (2 shared papers)Dong Hua (3 shared papers)Tingxun Lu (2 shared papers)Teng Wang (2 shared papers)Linfang Jin (2 shared papers)Hua Dong (1 shared paper)Pengfei Zhang (1 shared paper)Yun Chen (1 shared paper)
- Journals
- Journal of Surgical Oncology (2 papers)Oncology Reports (2 papers)Biochemical and Biophysical Research Communications (1 paper)Acta Pharmacologica Sinica (1 paper)European Journal of Medicinal Chemistry (1 paper)
- Partner nations
- ChinaUnited States
In The Last Decade
Kuan Ning
10 papers receiving 388 citations
Peers
Comparison fields: 5 of 64
- Cancer Research 190
- Sensory Systems 31
- Molecular Biology 282
- Oncology 70
- Biological Psychiatry 6
Countries citing papers authored by Kuan Ning
This map shows the geographic impact of Kuan Ning's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kuan Ning with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kuan Ning more than expected).
Fields of papers citing papers by Kuan Ning
This network shows the impact of papers produced by Kuan Ning. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kuan Ning. The network helps show where Kuan Ning may publish in the future.
Co-authors
The 25 scholars most cited alongside Kuan Ning, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 109 | |
| 2 | 2016 | 79 | |
| 3 | 2018 | 47 | |
| 4 | 2016 | 41 | |
| 5 | 2017 | 38 | |
| 6 | 2017 | 36 | |
| 7 | 2016 | 22 | |
| 8 | 2023 | 14 | |
| 9 | 2024 | 3 | |
| 10 | 2023 | 2 |
About Kuan Ning
Kuan Ning is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Oncology, Cancer Research and Surgery, having authored 10 papers that have together received 391 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (2 papers), Neuropeptides and Animal Physiology (2 papers), Extracellular vesicles in disease (2 papers), Stress Responses and Cortisol (1 paper), DNA Repair Mechanisms (1 paper), PARP inhibition in cancer therapy (1 paper), Cancer Genomics and Diagnostics (1 paper) and Cancer, Hypoxia, and Metabolism (1 paper). The work is most often cited by research in Cancer Research (190 citations), Sensory Systems (31 citations), Molecular Biology (282 citations), Oncology (70 citations) and Biological Psychiatry (6 citations). Kuan Ning has collaborated with scholars based in China and United States. Frequent co-authors include Jian Jin, Dong Hua, Tingxun Lu, Teng Wang, Linfang Jin, Hua Dong, Pengfei Zhang, Yun Chen, Dong Hua and Chun Zhang. Their work appears in journals such as Journal of Surgical Oncology, Oncology Reports, Biochemical and Biophysical Research Communications, Acta Pharmacologica Sinica and European Journal of Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.